Glossary for Movement symptoms

Medical terms related to Movement symptoms or mentioned in this section include:

• 10q Partial Trisomy: A very rare genetic disorder characterized by the duplication of genetic material from the long arm (q) of chromosome 10 - the genetic material occurs three times in body cells instead of the normal two. The type and severity of symptoms may vary considerably depending on the exact location and size of the duplicated genetic material.
• 11q Partial Trisomy: A very rare genetic disorder caused by a duplication of part of chromosome 11q. The characteristic symptoms of the disorder are delayed growth before and after birth, mental retardation (varying severity) and skull and facial defects. The type and severity of symptoms that can occur are variable.
• 1q deletion: A rare chromosomal disorder where part of the long arm (q) of chromosome 1 is deleted resulting in various abnormalities which are determined by the size of the deleted portion.
• 1q terminal deletion: A rare chromosomal disorder where the terminal part of the long arm (q) of chromosome 1 is deleted resulting in various abnormalities.
• 2-Hydroxyglutaricaciduria: A rare metabolic disorder characterized by high levels of a certain chemical (2-Hydroxyglutaric) which causes a serious progressive neurological disease and damage to the brain. The features of this disorder are variable and some cases are milder than others.
• 2-Methylbutyric Aciduria: A very rare genetic disorder where an enzyme deficiency prevents the break down of certain proteins into energy and results in a harmful accumulation of acids in the blood and body tissues. More specifically, there is a deficiency of an enzyme (2-methylbutyryl-coenzyme A dehydrogenase) needed to convert the amino acid isoleucine into energy. 2-methylbutyrylglycine levels build up in the body and may cause damage. Symptoms vary according to the degree of enzyme deficiency - can range from asymptomatic to life-threatening.
• 2-methylbutyryl-coenzyme A dehydrogenase deficiency: A very rare genetic disorder where an enzyme deficiency prevents the break down of certain proteins into energy and results in a harmful accumulation of acids in the blood and body tissues. More specifically, there is a deficiency of an enzyme (2-methylbutyryl-coenzyme A dehydrogenase) needed to convert the amino acid isoleucine into energy. 2-methylbutyrylglycine levels build up in the body and may cause damage. Symptoms vary according to the degree of enzyme deficiency - can range from asymptomatic to life-threatening.
• 22q11.2 deletion syndrome: A rare genetic disorder caused by the absence of a small portion of genetic material. A small section of chromosome 22 is missing at a location called q11.2. Chromosome 22 is one of 23 pairs of chromosomes that exist in humans.
• 2q deletion: A rare chromosomal disorder where part of the long arm (q) of chromosome 2 is deleted resulting in various abnormalities which are determined by the size of the deleted portion.
• 3 alpha methylglutaconicaciduria, type 3: A rare genetic condition where a gene mutation prevents the production of certain protein which leads to a build-up of an acid (3-methylglutaconic acid) which can have a negative impact on the body. The condition is characterized mainly by damage to the optic nerve.
• 3-Hydroxyacyl-CoA Dehydrogenase II Deficiency: A rare genetic disorder involving the deficiency of an enzyme (hydroxyacyl-coa dehydrogenase). The severity of the symptoms is highly variable with some cases resulting in death during the first decade while others suffer psychomotor and regression. Symptoms tend to be more severe in males who suffer progressive neurodegeneration whereas females tend to suffer mainly from developmental delay.
• 3-Hydroxyisobutyric aciduria: A rare inborn metabolic disorder which causes brain and facial anomalies, seizures and growth problems.
• 3-alpha-hydroxyisobutyryl-CoA hydrolase deficiency: A metabolic disorder involving an enzyme deficiency which causes symptoms such as degeneration of the nervous system. The other features of the disorder are somewhat variable.
• 3-methylglutaconic aciduria, type 1: A recessively inherited metabolic disorder characterized by methylglutaconic acid in the urine.
• 3-methylglutaconic aciduria, type 4: A rare genetic disorder where the body's cells are unable to make sufficient energy resulting in an accumulation in the body of 3-methylglutaconic acid. Type 4 is characterized by symptoms which overlap type 1 and 3.
• 3-methylglutaconic aciduria, type V: A rare genetic disorder where the body's cells are unable to make sufficient energy resulting in an accumulation in the body of 3-methylglutaconic acid.
• 3C syndrome: A rare disorder characterized by cardiac malformations, cerebellar hypoplasia and cranial dysmorphism which gives the disease it's name.
• 3q deletion: A rare chromosomal disorder where part of the long arm (q) of chromosome 3 is deleted resulting in various abnormalities which are determined by the size of the deleted portion.
• 4-hydroxyphenylacetic aciduria: A urinary abnormality usually caused by the deficiency of a particular enzyme (4-hydroxyphenylpyruvic acid oxidase). The urine contains excess 4-hydroxyphenylacetic acid.
• 46,XX chromosome 7 deletion p13-p21: A chromosomal disorder where a small portion of chromosome 7 is deleted which results in a range of abnormalities.
• 46,XY chromosome 7 deletion p13-p21: A chromosomal disorder where a small portion of chromosome 7 is deleted which results in a range of abnormalities.
• 47 XYY syndrome: A genetic condition where males have an extra Y chromosome in each of their cells. Normally male cells have one X and one Y chromosome. This is not usually an inherited condition but a defect that occurs during cell division. Often the condition is asymptomatic.
• 47,XXX syndrome: A genetic condition where females have an extra X chromosome in each of their cells. Normally female cells have two X chromosomes. This is not usually an inherited condition but a defect that occurs during cell division. Often the condition is asymptomatic.
• 49,XXXXY syndrome: A rare sex chromosome abnormality where there are three extra copies of the X chromosome.
• 4p16.3 deletion: A rare genetic disorder where a portion of chromosome 4 is deleted at a location called 16.3. The condition is characterized by malformations in most parts of the body as the deletion affects growth and development of the fetus.
• 6-pyruvoyl-tetrahydropterin synthase deficiency: A rare genetic disorder where insufficient levels of tetrahydropterin leads to a build up of phenylalanine in the blood which can cause toxic side effects such as nerve damage or even brain damage. The condition does not usually cause any significant symptoms.
• 7p2 Monosomy Syndrome: A rare chromosomal disorder where there is one copy of the end of the short arm (p) of chromosome 7 rather than the normal two. The type and severity of symptoms is determined by the location and size of the genetic material deleted.
• 7th cranial nerve palsy: 7th cranial nerve palsy, also called Bell's palsy, is a paralysis of the facial muscles on one side of the face due to unknown causes that affect the facial nerve.
• ACAD8 deficiency: An extremely rare metabolic disorder where the body is unable to metabolize certain proteins properly. More specifically, an insufficient level of the enzyme (isobutyryl-coenzyme A dehydrogenase) needed to metabolize the amino acid valine. The onset and severity of symptoms is variable.
• ACPS III: A rare genetic condition characterized by head and digital anomalies as well as other abnormalities.
• AChR deficiency and short channel open time: Extremely rare condition characterized by respiratory insufficiency from birth, facial dysplasia and paralysis of eye muscles.
• ADANE: A potentially fatal inherited neurological disease involving brain lesions. Symptoms tend to occur during childhood after an illness involving a fever. The disease is similar to Leigh syndrome but the course is acute rather than chronic.
• ADD: Attention Deficit Disorder (ADD) is a mental and behavioral disorder characterized by behavioral problems such as hyperactivity, inattention, concentration difficulty, and other mental symptoms. The related description Attention Deficit Hyperactivity Disorder (ADHD) may be a more modern description of the disease.

  Misdiagnosis of ADD is a well-known controversy in the sense that cases of hyperactivity in children may be over-diagnosed. There is a tendency for parents to seek and doctors to prescribe the drug Ritalin even in cases where the diagnosis of ADD or ADHD may be incorrect. Alternative diagnoses include normal child behavior (i.e. just an active child), food intolerances, or other behavioral disorders (see misdiagnosis of ADD).

  On the other hand, ADD is under-diagnosed in adults, with a large number of adults having ADD without knowing it; see misdiagnosis of Adult ADD.

• ADHD-like symptoms: is a neurodevelopmental behavioural disorder affecting 3-5% of the population characterised by 3 main components: hyperactivity, impulsivity and inattention. Some conditions which cause similar symptoms include
• ADHD-like symptoms in adults: is a neurodevelopmental behavioural disorder affecting 3-5% of the population characterised by 3 main components: hyperactivity, impulsivity and inattention. Some conditions which cause similar symptoms include
• ADHD-like symptoms in children: is a neurodevelopmental behavioural disorder affecting 3-5% of the population characterised by 3 main components: hyperactivity, impulsivity and inattention. Some conditions which cause similar symptoms include
• AIDS: A term given to HIV patients who have a low CD4 count (below 200) which means that they have low levels of a type of immune cell called T-cells. AIDS patients tend to develop opportunistic infections and cancers. Opportunistic infections are infections that would not normally affect a person with a healthy immune system. The HIV virus is a virus that attacks the body's immune system.
• ARCA: A group of recessively inherited neurological disorders characterized mainly by cerebellar ataxia and usually with other additional abnormalities.
• ARTS syndrome: A rare lethal syndrome characterized by deafness, optic atrophy and ataxia.
• ATR-X syndrome: A rare X-linked disorder that affects males and is characterized by mental retardation and alpha thalassemia.
• Aarskog Syndrome: A rare genetic condition characterized by facial, hand, genital and growth abnormalities.
• Abdominal Aneurysm: Dilatation of a section of the abdominal aorta, usually due to a weakness in the wall of the artery
• Abdominal muscle strain: Damage to the abdominal muscle due to over-stretching of the muscle tissue. The damage involves tearing the muscle tissue. Small blood vessels may also be damaged which can cause bruising. The symptoms may vary from mild to severe depending on the severity of the damage.
• Abdominal pain exacerbated by exercise: Any acute or chronic pathological condition of the abdomen can be exacerbated by physical exercise or a sporting activity. Some of the abdominal conditions which can be worsened by exercise or sports include
• Abdominal pain worsened by exercise: Abdominal pain in case of exercise is usually due to muscle cramps but previously present abdominal pain can be worsened in the following cases
• Abdominal swelling: Swelling or bloating of the abdomen
• Abductor lurch: deviation from the normal gait pattern expected for a child's age.
• Abetalipoproteinemia: A rare genetic disorder involving fat metabolism. The disorder is also known as Bassen-Kornzweig syndrome. Signs of the disease include acanthocytosis, little or no serum beta-lipoproteins and hypocholesterolemia. In severe cases, steatorrhea, ataxia, nystagmus, motor incoordination and retinitis pigmentosa may also occur.
• Abnormal Walk in Pregnancy: Abnormal Walk in Pregnancy is a change in the usual appearance of the way an adult woman moves when ambulating.
• Abnormal extensor reflex: also known as decerebrate posture
• Abnormal eye movements: Uncontrollable eye movements are involuntary, rapid, and repetitive movement of the eyes.
• Abnormal flexor response: An abnormal flexor response is an abnormal reflex to stimulation.
• Abnormal gait: An abnormal way of walking
• Abnormal gait in children: Abnormal gait in children refers to a type of walk that is irregular or deviates from the normal, possibly indicating pathology.
• Abnormal involuntary movements of the eyes: involuntary spasms of the eyelid muscles.
• Abnormal involuntary movements of the face: repetitive, spasmodic movement often involving the eyes and facial muscles.
• Abnormal involuntary movements of the mouth: repetitive spasmodic movements of the oral muscles
• Abnormal involuntary movements of the neck: repetitive spasmodic movements of the neck muscles.
• Abnormal involuntary movements of the tongue: repetitive spasmodic movements of the tongue.
• Abnormal movement during inspiration: decreased or increased movement of the chest wall during inspiration
• Abnormal walk: An abnormal walk is any type of gait that is irregular, unsteady, or unusual, possibly indicating pathology.
• Abscess: This is an area of puss collected in a cavity which is constituted by necrotised tissue
• Absence of septum pellucidum and septo-optic dysplasia: A rare birth defect where a thin membrane in the middle of the brain is missing. This brain abnormality is never present on it's own but is a characteristic of septo-optic dysplasia where the patient also has optic disk abnormalities and pituitary deficiencies.
• Absence of tibia: The congenital absence of the tibial bone which is the shin bone. One or both shin bones may be missing.
• Absence of tibia with polydactyly: The congenital absence of the tibial bone which is the shin bone as well as the presence of extra fingers.
• Absence seizure: An epileptic seizure involving momentary loss of consciousness.
• Absent abdominal reflexes: the abdominal reflex includes contraction of abdominal muscles in the quadrant of the abdomen that is stimulated by scraping the skin tangential to or toward the umbilicus. This contraction can often be seen as a brisk motion of the umbilicus toward the quadrant that is stimulated, it is absent in ceratin conditions
• Absent corneal reflex: Loss of blinking when the edge of the cornea is touched.
• Absent corneal reflex in one eye: Absent corneal reflex in one eye is the lack of a blink reflex in one eye.
• Absent corpus callosum -- cataract -- immunodeficiency: A rare syndrome characterized by immunodeficiency, cleft lip or palate, cataract, reduced pigmentation and brain abnormalities.
• Acanthamoeba infection: Infection with a microscopic, free-living ameba that is readily found in the environment - soil, air and water. Most people exposed to the ameba will not become infected but when infections do occur, they tend to affect the eyes, central nervous system or can cause widespread infection throughout the body.
• Acanthamoeba infection of the central nervous system: Infection by an amoebic organism called Acanthamoeba. Infection usually occurs when the amoeba enters through a break in the skin or through the nose. Infection can be localized or systemic where it can involve the central nervous system and cause potentially fatal meningoencephalitis. Infection of the eye can occur by cleaning contact lenses in contaminated water.
• Acanthocytosis: A rare disorder where most of the red blood cells are abnormal with spiny projections due to lipid abnormalities. The blood abnormality is seen in conditions such as abetalipoproteinemia, severe liver disease and severe malnutrition. Symptoms and prognosis depend on the underlying disorder.
• Accelerated hypertension: Accelerated hypertension is a condition characterized by a rapid increase in blood pressure. The condition is a medical emergency which can cause organ damage if not treated promptly.
• Aceruloplasminemia: A rare genetic disorder characterized by a lack of the protein ceruloplasmin in the blood resulting in a buildup of iron in the liver, brain and pancreas. This in turn causes diabetes and degeneration of the neural system causing tremors and walking abnormalities.
• Achalasia -- Addisonianism -- Alacrimia syndrome: A rare inherited disorder characterized mainly by achalasia, alacrimia (absent tears) and Addison's disease. Addison's disease involves adrenal insufficiency due to a resistance to adrenocorticotropic hormone. Only about 70 cases reported worldwide.
• Achalasia -- addisonianism -- alacrima syndrome: A rare inherited disorder characterized mainly by achalasia, alacrimia (absent tears) and Addison's disease. Addison's disease involves adrenal insufficiency due to a resistance to adrenocorticotropic hormone. Only about 70 cases reported worldwide.
• Achalasia -- adrenal -- alacrima syndrome: A familial disorder characterized by adrenal gland-related hormonal problems, swallowing difficulty (achalasia) and a lack of tears (alacrima). Neurological impairment and motor and sensory neuropathy is progressive. The adrenal glands in patients are resistant to the ACTH hormone and hence fails to operate normally.
• Achilles tendon bursitis due to running: It is estimated that Achilles tendonitis accounts for around 11% of all running injuries. The Achilles tendon is the large tendon at the back of the ankle. The achilles tendon can become inflamed through overuse as well as a number of contributory factors. The Achilles tendon has a poor blood supply which is why it is slow to heal.
• Achilles tendon stiff: tendon stiffness due to physical trauma or abnormal muscle contraction.
• Achrestic anemia: Achrestic anemia is a form of anemia similar to that caused by Vitamin B12 deficiency but it doesn't respond to treatment with Vitamin B12. The condition tends to progress slowly and can result in death if not treated. There are a variety of possible causes.
• Achromatopsia: Patients who have achromatopsia (sometimes called achromatopia) do not have normal "cone vision.". Although the term may refer to acquired disorders such as color agnosia and cerebral achromatopsia, it typically refers to an autosomal recessive congenital color vision disorder, the inability to perceive color AND to achieve satisfactory visual acuity at high light levels (typically exterior daylight).
• Acid-Base Imbalance: A disruption to the normal acid-base equilibrium in the body. There are four main groups of disorder involving an acid-base imbalance: respiratory acidosis or alkalosis and metabolic acidosis or alkalosis. Obviously the severity of symptoms is determined by the degree of imbalance.
• Acidemia, isovaleric: A rare genetic condition where the body can't process proteins adequately. More specifically, there are insufficient levels of the enzyme needed to break down an amino acid called leucine. This results in a build up of isovaleric acid which can harm the brain and nervous system. Some people suffer severe symptoms from birth and others suffer milder symptoms that come and go and are affected by such things as infections or consumption of high protein food.
• Acidemia, methylmalonic: An inborn error of metabolism where amino acids in the body aren't metabolized properly resulting in high levels of the acid throughout the body.
• Acidemia, propionic: An inherited genetic disorder where the body is incapable of processing some proteins and fats resulting in the accumulation of certain substances in the body which causes the symptoms of the condition. The condition can be life threatening.
• Acidic dry cell batteries inhalation poisoning: Acidic dry cell batteries contain toxic chemicals which can cause symptoms if inhaled. The smoke emitted from burning batteries can also cause poisoning symptoms if sufficient quantities are inhaled. The type and severity of symptoms varies depending on the amount of chemical involved.
• Ackee Fruit Food poisoning: Unripe ackee fruit contains a chemical called hypoglycin A and B which affect the central nervous system and fatty acid oxidation. Eating the unripe fruit can cause symptoms can occur in as little as two hours but is generally 6 to 48 hours. The ackee fruit is found mainly in Jamaica, West Africa, Central America, Sought Florida, southern California and Hawaii.
• Aconitase deficiency: A rare disorder where deficiency of an enzyme called aconitase results in muscle disease and intolerance to exercise.
• Acoustic Neurinoma: A benign tumor of the 8th cranial nerve which lies in the tube connecting the inner ear to the brain.
• Acoustic neuroma: A rare benign tumor that forms in the hearing canal. Can cause tinnitus, progressive hearing loss, headaches, facial numbness, papilledema, dizziness and an unsteady walk. Speaking and swallowing difficulty can occur in advanced stages. Also called acoustic neurilemoma, acoustic neurinoma and acoustic neurofibroma.
• Acquired Aplastic Anemia: A rare disorder involving severe failure of the bone marrow to produce new blood cells. Acquired aplastic anemia means that the condition was not present at birth but developed during the persons lifetime. The condition may be caused by such things as autoimmune reactions, radiation and certain drugs, chemicals or viral infections.
• Acquired Pure Red Cell Aplasia: An acquired condition which affects the formation of red blood cells and only red blood cells
• Acquired facial paralysis in children: Acquired facial paralysis in children is paralysis or the facial muscles in children that is due to a disease or cause that is not present at birth and is acquired later in life.
• Acquired hypothyroidism: Acquired hypothyroidism is a condition where the thyroid gland makes too little or no thyroid hormone. Acquired hypothyroidism can be caused by both thyroid disease (primary hypothyroidism) and hypothalamic-pituitary disease (central hypothyroidism)
• Acquired idiopathic sideroblastic anaemia: A rare disorder where iron is transported into a developing blood cells but because it is unable to be used, it builds up within the cell and tends to stop it from developing into a fully functioning red blood cell. Thus anemia can occur despite adequate or even high iron levels. Acquired cases can occur on exposure to excess alcohol, lead and drugs or can occur to nutritional problems involving a deficiency of folic acid or copper or an excess of zinc. The condition can also be caused by conditions such as kidney problems, endocrine dysfunction, metabolic disorders, rheumatoid arthritis and leukemia.
• Acrocallosal Syndrome (Schinzel Type): A rare condition characterized by absence of portion of the brain (corpus callosum), mental deficiency, duplicated toes, mental deficiency and other abnormalities.
• Acrocallosal syndrome: A rare genetic disorder characterized by underdeveloped or absent corpus callosum of brain, duplication of thumb or big toe and extra fingers or toes.
• Acrocephalopolydactyly -- Cardiac Disease -- Ear, Skin and Lower Limb Defects: A rare genetic condition characterized by head and digital anomalies as well as other abnormalities.
• Acrocephalopolysyndactyly type III: A rare genetic condition characterized by head and digital anomalies as well as other abnormalities.
• Acrodynia: A disease occurring in infants or young children. Symptoms include edema, pruritis, skin rash, extremities are pink, cheeks and nose are scarlet, profuse sweating, digestive disturbance, photophobia, polyneuritis, irritability, listlessness, apathy and failure to thrive.
• Acromegaloid hypertrichosis syndrome: A rare genetic condition characterized by excess body hair and a coarse face. The severity of the condition is variable.
• Acromegaly: An abnormal enlargement of the limbs due to increased secretion of growth hormone after the cessation of puberty
• Acromesomelic dysplasia Brahimi Bacha type: A very rare genetic malformation syndrome characterized primarily by developmental abnormalities of the face and skeletal bones.
• Acromesomelic dysplasia Hunter Thompson type: A rare genetic syndrome characterized by various severe developmental abnormalities of the skeletal bones.
• Acromesomelic dysplasia, Maroteaux type: A rare genetic syndrome characterized by various developmental abnormalities of the skeletal bones and facial anomalies.
• Acromicric dysplasia: A rare genetic syndrome characterized by various severe developmental abnormalities of the skeletal bones and facial anomalies.
• Acroosteolysis dominant type: A rare inherited connective tissue disorder characterized by breakdown of bone especially in the ends of the fingers and toes.
• Acroosteolysis neurogenic: A very rare inherited condition characterized mainly by the loss of all sensations - the lose the ability to feel pain, temperature and touch. The loss of sensation generally starts at the toes and fingers and spreads up the limbs and the trunk may also be involved in some cases.
• Actinomycetales infection: A bacterial infection from the order of Actinobacteria. The range of symptoms is variable depending on which bacteria from the order is involved.
• Acute Bokhoror: A brain disease caused by an unknown pathogen which is probably from the Picornavirus family of viruses. Mode of transmission is uncertain but genetic susceptibility may be involved. The incubation period appears to be an average of 15 years. The disease can be classified according to rate of progression: acute or subacute, slowly progressive and chronic. Death is common in the acute phase of the infection which can last from four days to four months.
• Acute Chemical poisoning -- Varnish makers' and painters' Naptha: Varnish makers' and painters' Naptha is an ingredient used in certain pesticides. Exposure to the chemical can cause a range of symptoms depending on the level and route of exposure. Exposure can occur through inhalation, ingestion, the skin or eyes. Acute exposure involves a exposure over a short period of time whereas chronic exposure occurs over a longer period of time.
• Acute Disseminated Encephalomyelitis: A type of encephalitis that usually follows an acute viral infection and involves an immune attack on myelin tissue which is part of the nervous system. Initial symptoms include fever, headache, vomiting and drowsiness followed by seizures, coma and paralysis. Often results in permanent neurological disorders.
• Acute Pesticide poisoning -- Triforine: Triforine is an ingredient used in certain herbicides. Exposure to the chemical can cause a range of symptoms depending on the level and route of exposure. Exposure can occur through inhalation, ingestion, the skin or eyes. Acute exposure involves a exposure over a short period of time whereas chronic exposure occurs over a longer period of time.
• Acute Pesticide poisoning -- xylene: Xylene is an ingredient used in certain insecticides. Exposure to the chemical can cause a range of symptoms depending on the level and route of exposure. Exposure can occur through inhalation, ingestion, the skin or eyes. Acute exposure involves a exposure over a short period of time whereas chronic exposure occurs over a longer period of time.
• Acute Silicosis: An occupation lung disease caused by breathing in high levels of silica dust.
• Acute VE: A brain disease caused by an unknown pathogen which is probably from the Picornavirus family of viruses. Mode of transmission is uncertain but genetic susceptibility may be involved. The incubation period appears to be an average of 15 years. The disease can be classified according to rate of progression: acute or subacute, slowly progressive and chronic. Death is common in the acute phase of the infection which can last from four days to four months.
• Acute Viliuisk Encephalitis: A brain disease caused by an unknown pathogen which is probably from the Picornavirus family of viruses. Mode of transmission is uncertain but genetic susceptibility may be involved. The incubation period appears to be an average of 15 years. The disease can be classified according to rate of progression: acute or subacute, slowly progressive and chronic. Death is common in the acute phase of the infection which can last from four days to four months.
• Acute Viliuisk Encephalomyelitis: A brain disease caused by an unknown pathogen which is probably from the Picornavirus family of viruses. Mode of transmission is uncertain but genetic susceptibility may be involved. The incubation period appears to be an average of 15 years. The disease can be classified according to rate of progression: acute or subacute, slowly progressive and chronic. Death is common in the acute phase of the infection which can last from four days to four months.
• Acute Vilyuisk Encephalitis: A brain disease caused by an unknown pathogen which is probably from the Picornavirus family of viruses. Mode of transmission is uncertain but genetic susceptibility may be involved. The incubation period appears to be an average of 15 years. The disease can be classified according to rate of progression: acute or subacute, slowly progressive and chronic. Death is common in the acute phase of the infection which can last from four days to four months.
• Acute Vilyuisk Encephalomyelitis: A brain disease caused by an unknown pathogen which is probably from the Picornavirus family of viruses. Mode of transmission is uncertain but genetic susceptibility may be involved. The incubation period appears to be an average of 15 years. The disease can be classified according to rate of progression: acute or subacute, slowly progressive and chronic. Death is common in the acute phase of the infection which can last from four days to four months.
• Acute adhd-like symptoms: is a neurodevelopmental behavioural disorder affecting 3-5% of the population characterised by 3 main components: hyperactivity, impulsivity and inattention. Some conditions which cause similar symptoms include
• Acute adhd-like symptoms in adults: is a neurodevelopmental behavioural disorder affecting 3-5% of the population characterised by 3 main components: hyperactivity, impulsivity and inattention. Some conditions which cause similar symptoms include
• Acute adhd-like symptoms in children: is a neurodevelopmental behavioural disorder affecting 3-5% of the population characterised by 3 main components: hyperactivity, impulsivity and inattention. Some conditions which cause similar symptoms include
• Acute cholinergic dysautonomia: A rare condition characterized by the presence of abnormal red blood cells in the bone marrow and blood. The condition is characterized by anemia and generally leads to the development of acute myelogenous leukemia. The acute form has more severe symptoms than the chronic form.
• Acute episodic adhd-like symptoms: ADHD is a neurodevelopmental behavioural disorder affecting 3-5% of the population characterised by 3 main components: hyperactivity, impulsivity and inattention. Some conditions which cause similar symptoms but episodic in nature include
• Acute fulminant multiple sclerosis: Malignant Multiple Sclerosis, is a particularly aggressive form of the disease. Thankfully very rare, this highly aggressive form is defined by its swift and relentless decline to significant disability or even death, often within a few weeks or months after the onset of the initial attack. It is characterized by widespread and progressive cerebral white matter destruction or by severe pathological involvement of clinically strategic regions such as brainstem, resulting in bulbar paralysis.
• Acute headache: Headache, or cephalgia, is defined as diffuse pain in various parts of the head, with the pain not confined to the area of distribution of a nerve.
• Acute heartburn after exercise: exercise maybe be a precipitating factor to some conditions presenting with heartburn
• Acute hyperactivity in adults: maybe to due to various causative agents
• Acute hyperactivity in the workplace: maybe due to stress or an underlying neurological lesion
• Acute idiopathic polyneuritis: An inflammatory disorder of the peripheral nerves The condition is characterized by weakness, numbness or tingling in the legs or arms or occasionally loss of movement and feeling in the legs, arms, upper body and face. Some patients have minor symptoms and others suffer severe symptoms such as paralysis. Also called Guillain-Barre syndrome, infectious polyneuritis or acute febrile polyneuritis.
• Acute injuries of both knees related to sports: Acute injuries of both knees related to sports is the sudden onset of sports-related trauma to the knees.
• Acute injuries of the knee related to sports: The knee joint is the largest joint in the body, consisting of 4 bones and an extensive network of ligaments and muscles. Injuries to the knee joint are amongst the most common in sporting activities.
• Acute intermittent porphyria: A rare inherited metabolic disorder caused by a disturbed porphyrin metabolism resulting in increased production of porphyrin or its precursors. Symptoms include abdominal pain, photosensitivity and neurological disturbances such as seizures, coma, hallucinations and respiratory paralysis.
• Acute leukaemia of ambiguous lineage: A term used to describe a type of leukemia (a blood cancer) where the leukemic cells cannot be determined as myeloid or lymphoid or where both types of cells are present.
• Acute lymphoblastic leukemia: A malignant disease that starts suddenly and progresses quickly. It is characterized by a high number of immature cells in the organs, bone marrow and blood. Symptoms include fever, pallor, anorexia, fatigue, anemia, hemorrhage, bone pain, splenomegaly and frequent infections. Also called acute lymphocytic leukemia.
• Acute lymphoblastic leukemia, Susceptibility to: Cancer of the white blood cells characterized by the presence of excessive lymphoblasts. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets. There are two subtypes of leukemia linked to a genetic anomaly which increases a person's susceptibility to developing the cancer. Type 1 is linked to a defect on chromosome 10q21 and type 2 is linked to a defect on chromosome 7p12.2.
• Acute lymphoblastic leukemia, Susceptibility to, 1: Cancer of the white blood cells characterized by the presence of excessive lymphoblasts. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets. There are two subtypes of leukemia linked to a genetic anomaly which increases a person's susceptibility to developing the cancer. Type 1 is linked to a defect on chromosome 10q21.
• Acute lymphoblastic leukemia, Susceptibility to, 2: Cancer of the white blood cells characterized by the presence of excessive lymphoblasts. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets. There are two subtypes of leukemia linked to a genetic anomaly which increases a person's susceptibility to developing the cancer. Type 2 is linked to a defect on chromosome 7p12.2.
• Acute lymphoblastic leukemia, adult: Cancer of the white blood cells. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets.
• Acute megacaryoblastic leukemia: A rare form of malignant bone marrow cancer involving the proliferation of immature precursors of blood cells. More specifically, it involves the rapid proliferation of megakaryoblasts (premature form of megakaryocytes).
• Acute meningitis: Acute meningitis is an inflammation of the brain that presents in an acute fashion. The inflammation may be the result of infective agents such as bacteria, viruses and fungi as well as non-infective agents such as certain drugs. Acute forms of meningitis can develop in within hours or days whereas chronic meningitis develops over weeks or months.
• Acute myeloblastic leukemia type 1: A form of blood cancer resulting in the rapid proliferation of immature blood cells (blast cells).
• Acute myeloblastic leukemia type 2: A form of blood cancer resulting in the rapid proliferation of granulocytes and monocytes.
• Acute myeloblastic leukemia type 3: A rare form of malignant bone marrow cancer involving the rapid proliferation of immature precursors of blood cells. Type 3 involves the proliferation of promyelocytes.
• Acute myeloblastic leukemia type 4: A rare form of malignant bone marrow cancer involving the rapid proliferation of immature precursors of blood cells. Type 4 involves the rapid proliferation of myelocytes and monocytes.
• Acute myeloblastic leukemia type 5: A rare form of malignant bone marrow cancer involving the proliferation of immature precursors of blood cells. Type 5 involves the rapid proliferation of monoblasts (immature precursors of monocytes) in particular.
• Acute myeloblastic leukemia type 6: A rare form of malignant bone marrow cancer involving the rapid proliferation of immature precursors of blood cells. Type 6 involves the proliferation of the immature precursors of red blood cells called erythroblasts.
• Acute myeloblastic leukemia type 7: A rare form of malignant bone marrow cancer involving the proliferation of immature precursors of blood cells. Type 7 involves the rapid proliferation of megakaryoblasts (premature form of megakaryocytes) in particular.
• Acute myelocytic leukemia: A malignant cancer of blood-forming tissues resulting in a high number of immature leukocytes. Symptoms include soft bleeding gums, anemia, fatigue, fever, dyspnea, moderate splenomegaly, joint and bone pains and frequent infections. Also called acute granulocytic leukemia, acute myelogenous leukemia, acute nonlymphocytic leukemia, myeloid leukemia, splenomedullary leukemia, splenomyelogenous leukemia.
• Acute myelofibrosis: A rare condition where progressive scarring or fibrosis of the bone marrow impairs it's ability to make blood cells causing symptoms such as anemia and liver and spleen enlargement.
• Acute myeloid leukaemia and myelodysplastic syndromes related to alkylating agent: The use of alkylating agents to treat cancer can result in leukemia in some patients.
• Acute myeloid leukaemia and myelodysplastic syndromes related to topoisomerase type II inhibitor: The use of topoisomerase type II inhibitors to treat cancer can result in leukemia in some patients.
• Acute myeloid leukaemia and myelodysplastic syndromes, therapy related: Certain cancer therapies can result in the development of leukemia in some patients. These therapies includes topoisomerase type II inhibitors and alkylating agents.
• Acute myeloid leukemia: A form of rapidly progressing blood cancer resulting in the rapid proliferation of granulocytes and monocytes, red blood cells and platelets.
• Acute myeloid leukemia, adult: A form of blood cancer resulting in the rapid proliferation of granulocytes and monocytes, red blood cells and platelets.
• Acute myelosclerosis: A rare disorder where the bone marrow makes too many blood cells. The disease progresses rapidly with death usually occurring within 6 months of onset.
• Acute non lymphoblastic leukemia: A form of rapidly progressing blood cancer resulting in the rapid proliferation of granulocytes and monocytes, red blood cells and platelets. It is one of the most common forms of leukemia in adults but can occur in children.
• Acute pain sitting down: rest pain occurs when blood flow in the extremity falls below resting tissue requirements
• Acute pain when walking: also known as intermittent claudication
• Acute pain when walking in pregnancy: Acute pain when walking in pregnancy refers to pain in the region of the hips, lower back and pelvis, occurring in the ambulant woman during pregnancy and often increasing with gestation.
• Acute panmyelosis: A rare condition where progressive scarring or fibrosis of the bone marrow impairs it's ability to make blood cells causing symptoms such as anemia and liver and spleen enlargement. The disease often progresses rapidly and results in death.
• Acute rheumatic fever: Bacterial joint infection with risk of heart complications.
• Acute sinusitis: An acute inflammation of the sinuses
• Acute vitamin A toxicity: Acute ingestion of vitamin A can cause symptoms. Symptoms usually only last for a day or two.
• Acyl-CoA dehydrogenase, short chain, deficiency of: A rare disorder where the body lacks enzymes needed to convert some fats (short-chain fatty acids) into energy. Symptoms are exacerbated by fasting or acute illness. The severity of symptoms is variable with some patients remaining virtually asymptomatic their whole life while other suffer symptoms from infancy.
• Acyl-CoA dehydrogenase, very long chain, deficiency of: A rare inherited genetic condition where the body is unable to convert certain fats to energy i.e. there is not enough of a certain enzyme which is needed to metabolize a type of fat called long-chain fatty acids. The build-up of these fatty acids in the body causes damage. There are three subtypes of the disorder each with varying severity: severe early-onset form, an intermediate form and an adult-onset form.
• Adam and Eve poisoning: The Adam and Eve plant is a herb with heart-shaped leaves found in Europe. The plant contains a poisonous chemical called calcium oxalate crystals which can cause a variety of symptoms if ingested. Eye exposure can also cause symptoms due to the abrasive nature of the toxic chemical. Ingestion of the plant generally causes severe mouth pain. Skin exposure usually only causes minor, short-lived skin irritation.
• Adamantinoma: A very aggressive malignant cancer of the jaw. Also called ameloblastoma, adamantoblastoma or epithelioma adamantinum.
• Adams Nance syndrome: A rare genetic disorder characterized by rapid heartbeat, high blood pressure, small eyes and the presence of excess glycine in the urine.
• Addiction symptoms: Symptoms related to addiction (physical or mental addiction)
• Adducted thumb syndrome recessive form: A rare recessively inherited disorder characterized mainly by a small head, arthrogryposis (joint contractures), cleft palate and various other abnormalities.
• Adducted thumbs -- arthrogryposis, Christian type: A rare recessively inherited disorder characterized mainly by a small head, arthrogryposis (joint contractures), cleft palate and various other abnormalities.
• Adduction and extension of the arms: position of the upper limb due various etiologies
• Adduction of the arms: position of the upper limb due various etiologies
• Adenocarcinoma of lung: A tumor that develops in the lining of the lung. The tumor is usually slow growing.
• Adenocarcinoma, Bronchiolo-Alveolar: A form of lung cancer that develops in the bronchioles or alveoli.
• Adenoid cystic carcinoma: A malignant cancer in the form of cysts which may occur in the salivary glands, breast, mucous glands of the respiratory tract and sometimes in vulval vestibular glands. Also called adeoncystic carcinoma, adenomyoepithelioma, cribriform carcinoma or cylindroma.
• Adenosarcoma of the uterus: A tumor that develops from the glands that line the uterus.
• Adenosine triphosphatase deficiency, anaemia due to: Deficiency of a chemical (adenosine triphosphate) resulting in anemia.
• Adenylosuccinate lyase deficiency: A rare inherited disorder characterized by a deficiency of the enzyme called adenlyosuccinate lyase which generally results in psychomotor retardation and autistic behavior.
• Adhd: Attention Deficit Hyperactivity Disorder (ADHD) is a mental and behavioral disorder characterized by behavioral problems such as hyperactivity, inattention, concentration difficulty, and other mental symptoms. Typically, ADHD and associated hyperactivity is known as a childhood disorder, although ADD/ADHD in adults is known to be under-diagnosed. It is distinguished from Attention Deficit Disorder (ADD) which has a reduced focus on hyperactivity type symptoms.
• Adhesive abuse: Adhesive abuse is the use of various inhalants for the purpose of achieving a "high". They are often used as a cheap, readily available alternative to street drugs but they can cause serious damage to the body. Adhesives include household glues, rubber cement and model aeroplane glue. These adhesives can be abused by sniffing them, spraying directly into the mouth, heating them and then inhaling them or injecting them directly into the body.
• Adhesive addiction: Adhesive addiction refers to the compulsive need to abuse adhesives (e.g. sniffing them). Sufferers have withdrawal symptoms when attempting to stop the habit and feel unable to stop the habit despite knowing the harm it is causing their health. Aerosols are very damaging to the body and can readily result permanent brain damage and even death. Death can occur through chronic use and in rare cases can occur after one session of use. Children and teenagers are particular at risk for this type of addiction - it is readily available and users feel it gains them greater acceptance from their peers. Adhesives includes household glue, rubber cement and model airplane glue.
• Adrenal Cancer: A malignant cancer that develops in the adrenal gland. The tumor may be nonfunctioning (does not produce hormones) or functioning in which case excessive levels of hormones can cause a variety of symptoms depending on which hormone is involved. Adrenal hormones made in the cortex (outer part of the gland) are aldosterone, corticosteroids and androgenic steroids. Adrenalin and noradrenalin are the hormones made in the medulla (central part of the adrenal gland).
• Adrenal Cortex Diseases: Diseases of the adrenal cortex. Examples includes Addison's disease, Cushing's syndrome and adrenal fatigue.
• Adrenal Cortex Neoplasms: A tumor that develops in the adrenal gland. The tumor may be nonfunctioning (does not produce hormones) or functioning in which case excessive levels of hormones can cause a variety of symptoms depending on which hormone is involved. Adrenal hormones made in the cortex (outer part of the gland) are aldosterone, corticosteroids and androgenic steroids.
• Adrenal adenoma, familial: A benign tumor that develops in the adrenal gland and tends to run in families. The tumor may be nonfunctioning (does not produce hormones) or functioning in which case excessive levels of hormones can cause a variety of symptoms depending on which hormone is involved. Adrenal hormones made in the cortex (outer part of the gland) are aldosterone, corticosteroids and androgenic steroids . Adrenalin and noradrenalin are the hormones made in the medulla (central part of the adrenal gland).
• Adrenal crisis: A potentially fatal condition where the adrenal cortex slows or stops functioning resulting in reduced glucocorticoids, decreased extracellular fluid volume and hyperkalemia. Symptoms include shock, coma, low blood pressure, weakness and loss of vasomotor tone. Also called addisonian crisis.
• Adrenal disorders: Disorders affecting the adrenal glands
• Adrenal gland hyperfunction: Excessive activity of the adrenal gland which causes excessive production of one or more adrenal hormones (aldosterone, corticosteroids, androgenic steroids, epinephrine and norepinephrine). The increased adrenal gland activity may be caused by an adrenal gland tumor or by excessive stimulation of the gland. Pituitary hormones stimulate adrenal gland activity.
• Adrenal gland hypofunction: Reduced adrenal gland activity due to damage to the adrenal gland or lack of stimulation of the gland. Pituitary hormones stimulate adrenal gland activity.
• Adrenal gland symptoms: Symptoms affecting the adrenal glands
• Adrenal hyperplasia: A group of disorder that occur when there is a problem in the process of making adrenal corticosteroids.
• Adrenal hypertension: Adrenal hypertension is high blood pressure caused by adrenal gland problems. For example, an adrenal tumor can cause excessive production of aldosterone which in turn causes salt-retention and high blood pressure. Severity of symptoms varies depending on the underlying cause.
• Adrenal hypofunction: A condition which is characterized by a lack of production of hormones from the adrenal gland.
• Adrenal incidentaloma: A tumor of the adrenal gland that is discovered incidentally while performing an imaging examination for reasons other than an adrenal tumor. The tumor may be asymptomatic or can causes excessive secretion of adrenal hormones and resulting symptoms. The tumor may also be malignant or benign.
• Adrenal medulla neoplasm: A tumor that develops in the part of the adrenal gland called the medulla which produces adrenalin and noradrenaline. The tumor is usually benign but can be malignant.
• Adrenocortical carcinoma: A condition which is characterized by malignancy which affects the adrenocortex.
• Adrenoleukodystrophy: A rare hereditary metabolic disease that only occurs in male children and is characterized by adrenal atrophy and extensive cerebral demyelination causing progressive loss of mental functioning, aphasia, apraxia and sometimes blindness. The patient usually dies within 5 years.
• Adrenoleukodystrophy, autosomal, neonatal form: A rare inherited disorder involving the adrenal glands, testes and certain parts of the brain (white matter). It is a less severe form of leukodystrophy where an abnormality within the body cells prevents the metabolism of certain fats (long chain fatty acids).
• Adrenomyeloneuropathy: A form of X-linked adrenoleukodystrophy characterized by spinal cord dysfunction and brain involvement may or may not be present. Those with brain involvement suffer serious symptoms that can eventually lead to total disability and even death.
• Adult ADHD: Adult ADHD, (attention deficit hyperactivity disorder) is a common neurobehavioral developmental disorder with an onset in childhood that continues into adulthood. Children do not simply grow out of ADHD, as is often believed. Just the opposite is commonly true - the symptoms of ADHD often get worse as a child grows into adulthood. The predominant behaviors of adult ADHD are the same as in children and include:
  • Inattentiveness
  • Hyperactivity
  • Impulsivity

  These behaviors result in difficulties with:

  • Concentration
  • Remaining focused on a task or activity
  • Controlling behavior
  • Hyperactivity or over-activity

  The symptoms of adult ADHD can be treated, but there currently is no cure for the disorder. Most people with ADHD can be successfully treated and lead normal, productive lives at home, work, school and with friends and family. The cause or causes of ADHD are not yet known, although researchers believe that genes may be one factor in the development of the disease. It is most likely that the disorder is the result of a combination of elements, including environmental factors, traumatic head injuries, nutrition, and social influences.

• Adult Panic-Anxiety Syndrome: A psychiatric disorder involving anxiety and panic attacks that occur for no obvious reason.
• Adult SMA: Form of Spinal Muscular Atrophy in adults.
• Adult low grade infiltrative supratentorial Astrocytoma: A type of brain cancer that occurs in the supratentorial region of the brain of adults and is relatively non-aggressive.
• Adult progressive spinal muscular atrophy, Aran Duchenne type: A group of inherited motor neuron diseases involving progressive muscle weakness, wasting and paralysis due to degeneration of motor neurons in the spinal cord. Muscle weakness and wasting usually starts in the hands and may gradually spread to other muscle groups.
• Adult-onset ALD: Form of ALD in adults.
• Adult-onset asthma: Adult-onset asthma is a type of asthma that occurs during adulthood. Asthma involves constriction and inflammation of the airways. The severity of symptoms is variable. Allergies account for about half the cases of adult-onset asthma.
• Aerosol abuse: Aerosol abuse is the use of various inhalants for the purpose of achieving a "high". They are often used as a cheap, readily available alternative to street drugs but they can cause serious damage to the body. Aerosols include air fresheners, hair spray, spray pain and deodorants. These aerosols can be abused by sniffing them, spraying directly into the mouth, heating them and then inhaling them or injecting them directly into the body.
• Aerosol addiction: Aerosol addiction refers to the compulsive need to abuse aerosol (e.g. sniffing them). Sufferers have withdrawal symptoms when attempting to stop the habit and feel unable to stop the habit despite knowing the harm it is causing their health. Aerosols are very damaging to the body and can readily result permanent brain damage and even death. Death can occur through chronic use and in rare cases can occur after one session of use. Children and teenagers are particular at risk for this type of addiction - it is readily available and users feel it gains them greater acceptance from their peers. Aerosols includes spray pain, air freshener, deodorants and hair sprays.
• Aerotitis syndrome: Trauma to the blood vessels in the ears caused by rapid changes in atmospheric pressure. Blockage of the Eustachian tube in the ear prevents equalization of air pressure and a vacuum develops inside the ear. Yawning or chewing can sometimes alleviate symptoms by opening up the Eustachian tube.
• African Sleeping sickness: A disease caused by parasites (Trypanosome brucei gamiense or T. brucei rodesiense) and transmitted to humans by the tsetse fly which is found only in Africa. Causes symptoms such as fever, chills, headache, anemia, edema of hands and feet, enlarged lymph glands, lethargy, sleepiness, convulsions and coma. Also called African trypanosomiasis and sleeping sickness.
• Agenesis of the corpus callosum: Congenital absence of connective part of the brain.
• Aggressive fibromatosis -- parapharyngeal space: A type of tumor that occur near in the space around the pharynx and is locally invasive but not malignant. They tend to occur mainly in the head and neck region and symptoms depend on the exact location and aggressiveness of the tumor. Tumors often reoccur after surgical removal which then requires further treatment with radiation and chemotherapy.
• Aging brain syndrome: Aging processes in the brain can cause various psychological and neurological symptoms.
• Agitation: A state of increased tension with episodes of emotional and physical irritability.
• Agnathia-holoprosencephaly-situs inversus: A very rare disorder characterized by a small or absent jaw, developmental brain defect and internal organs situated on the wrong side of the body (situs inversus). The severity and range of symptoms is variable.
• Agyria: Abnormal condition where the body excessively absorbs silver salts and deposits it in the tissues. Symptoms include gray skin and mucous membranes.
• Agyria-pachygyria type 1: Abnormal brain development where the brain fails to develop normally during the fetal stage.
• Aicardi syndrome: A rare genetic disorder where the structure connecting the two halves of the brain fails to develop which results in seizures and eye abnormalities .
• Aicardi-Goutieres syndrome: A rare inherited progressive disease that affects the brain and immune system.
• Aicardi-Goutieres syndrome 1: A rare inherited progressive disease that affects the brain and immune system. Type 1 is caused by a defect on chromosome 3p21.3-p21.2.
• Aicardi-Goutieres syndrome 2: A rare inherited progressive disease that affects the brain and immune system. Type 2 is caused by a defect on chromosome 13q14-q21.
• Aicardi-Goutieres syndrome 3: A rare inherited progressive disease that affects the brain and immune system. Type 3 is caused by a defect on chromosome 11q13.2.
• Aicardi-Goutieres syndrome 4: A rare inherited progressive disease that affects the brain and immune system. Type 4 is caused by a defect on chromosome 19p13.13.
• Aicardi-Goutieres syndrome 5: A rare inherited progressive disease that affects the brain and immune system. Type 5 is caused by a defect on chromosome 3p21.3-p21.2.
• Air embolism: A condition where an air bubble enters the cardiovascular system (via injection, intravenous therapy, surgery or puncture wound) and obstructs the blood flow.
• Air sickness: disturbance between the central nervous system and the inner ear which affects the balance and equilibrium
• Akathisia: A condition characterized by a constant urge to move resulting in the sufferer being unable to sit still. Can be caused by use of anti-psychotic drugs or anti-depressants or can occur spontaneously.
• Akathisia in children:
• Akesson syndrome: A very rare syndrome characterized by excessive skin folds and furrows on the scalp, mental retardation the failure of the thyroid to develop.
• Al Gazali -- Nair syndrome: A very rare syndrome characterized by bone abnormalities, eye problems, seizures and developmental delay. The reported cases involved related parents.
• Al Gazali Sabrinathan Nair syndrome: A very rare syndrome characterized by bone and eye problems, seizures and mental retardation.
• Al Murrah-induced lead poisoning: Al Murrah is a folk remedy used mainly by Saudi Arabian people to treat problems such as stomach pain, diarrhea and colic. This folk remedy has the potential to cause lead poisoning due to its relatively high content of lead. Children are more susceptible to the effects of lead. The use of folk remedies is still prevalent in some cultures. Lead poisoning can result in serious illness and even death in severe cases.
• Alagille syndrome: A genetic disorder affecting the liver and characterized by the absence of some or all of the liver bile ducts that transport bile within the liver.
• Alajouanine syndrome: A birth disorder characterized mainly by clubfoot, strabismus and facial paralysis. The facial paralysis is caused by damage to the 6th and 7th cranial nerve.
• Alarcon-induced lead poisoning: Alarcon is a folk remedy used mainly by Mexican people to treat digestive or stomach problems including indigestion and diarrhea. This folk remedy has the potential to cause lead poisoning due to its relatively high content of lead. Children are more susceptible to the effects of lead. The use of folk remedies is still prevalent in some cultures. Lead poisoning can result in serious illness and even death in severe cases.
• Albayaidle-induced lead poisoning: Albayaidle is a folk remedy used mainly by Mexican and Central American people to treat digestive or stomach problems such as vomiting and colic. It is also used to treat apathy and lethargy. This folk remedy has the potential to cause lead poisoning due to its relatively high content of lead. Children are more susceptible to the effects of lead. The use of folk remedies is still prevalent in some cultures. Lead poisoning can result in serious illness and even death in severe cases.
• Albayalde-induced lead poisoning: Albayalde is a folk remedy used mainly by Mexican and Central American people to treat digestive or stomach problems such as vomiting and colic. It is also used to treat apathy and lethargy. This folk remedy has the potential to cause lead poisoning due to its relatively high content of lead. Children are more susceptible to the effects of lead. The use of folk remedies is still prevalent in some cultures. Lead poisoning can result in serious illness and even death in severe cases.
• Albers-Schonberg disease -- Adult benign dominant form: A rare disorder characterized primarily by increased bone density as old bone is not resorbed and replaced with new bone - is also known as marble bone disease. The adult benign form is associated with a normal life expectancy and is often asymptomatic.
• Albers-Schonberg disease -- intermediate form: A rare disorder characterized primarily by increased bone density as old bone is not resorbed and replaced with new bone - is also known as marble bone disease. The intermediate form is more severe than the adult form but less severe than the infantile form. Life expectancy is usually normal.
• Albright's hereditary osteodystrophy: A rare genetic disorder where the body fails to recognize and respond to the parathyroid hormone. The parathyroid hormone is involved in controlling the blood levels of calcium and phosphate.
• Alcohol Withdrawal: Symptoms that occur when alcohol consumption is discontinued or reduced. Symptoms may vary depending on the level of dependence.
• Alcohol abuse: Excessive alcohol as a symptom of other conditions
• Alcohol drinking: The consumption of a drink containing alcohol. Alcohol consumption can cause varying degrees of impairment depending on the amount consumed. Consuming very large amounts of alcohol can lead to death.
• Alcohol-Induced Disorders: Disorders caused by excessive alcohol consumption. The symptoms are variable depending on the disorder involved. Some of the disorders are: alcohol abuse, alcohol dependence, alcohol intoxication, alcohol withdrawal, alcohol intoxication delirium, alcohol withdrawal delirium, alcohol-induced persisting dementia, alcohol-induced persisting amnestic disorder, alcohol-induced psychotic disorder, alcohol-induced mood disorder, alcohol-induced anxiety disorder, alcohol-induced sexual dysfunction, alcohol-induced sleep disorder, liver damage, liver cancer and esophageal cancer.
• Alcohol-induced hypertension: Alcohol-induced hypertension is high blood pressure caused by excessive drinking of alcohol.
• Alcoholic Neuropathy: Neurological changes due to nerve damage from long-term alcohol consumption
• Alcoholic cerebellar degeneration: cerebellar degeneration which occurs in alcoholics
• Alcoholic intoxication: The excessive consumption of alcohol can have toxic effects on the body and can ultimately result in death in severe cases.
• Alcoholic polyneuropathy: A condition where damage to many peripheral nerves throughout the body results from excessive alcohol consumption. The sensory nerves tend to be affected more than the motor nerves and the legs are usually more affected than the arms.
• Alcoholic, reversible acute muscular: Muscle cramps associated with chronic alcohol abuse.
• Aldolase A deficiency: A rare condition where a deficiency of the enzyme called aldolase A causes muscle problems and anemia.
• Alertness: A state of function in which someone is watchfull
• Alexander Syndrome: Brain myelin disorder causing mental degeneration.
• Alkalosis: A condition that iscaused by the accumulation of base in the body.
• Alkaptonuria: A rare inherited metabolic disease characterized by homogentisic aciduria, arthritis and ochronosis. Symptoms include darkening of urine, alkinization due to overproduction of homogentisic acid, arthritis in the large joints and black ochronotic pigmentation of cartilage and collagen tissue. However, many of these symptoms may not occur until middle age. The condition may also be caused by chronic phenol poisoning.
• Allain Babin Demarquez syndrome: A rare syndrome characterized by premature fusion of skullbones, abnormal development of skeletal bones and hypertension.
• Allan-Herndon-Dudley Syndrome: A very rare inherited disorder characterized primarilty by mental retardation.
• Allergenic cross-reactivity: Studies have indicated that a significant number of people with certain allergies will also have allergic responses to other allergens which have a similar protein. For example patients allergic to birch pollen will often have allergies to plant foods such as apples and peaches. Symptoms can range from mild response to severe allergic reactions. Cross-reactivity tends to have mainly oral allergy symptoms with breathing problems and anaphylactic reactions being extremely rare. Food allergies related to cross-reactivity tend to be less severe than those not related to cross-reactivity.
• Allergic encephalomyelitis: An autoimmune brain and spinal cord disease that can be induced in laboratory animals in experimental settings. The disease involves inflammation and degeneration of nerve myelin sheaths and it may be acute or chronic.
• Allergic irritability syndrome: Symptoms such as irritability and poor concentration associated with nose, ear and sinus symptoms caused by allergies such as hayfever.
• Allergic tension-fatigue syndrome: Variable symptoms caused by food allergy.
• Alopecia -- hypogonadism -- extrapyramidal disorder: A rare syndrome characterized by alopecia, progressive movement problems and a lack of gonadal function which affects puberty.
• Alopecia mental retardation syndrome: A rare syndrome characterized primarily by a lack of hair and mental retardation.
• Alopecia, epilepsy, oligophrenia syndrome of Moynahan: A rare condition characterized by alopecia, epilepsy, mental retardation and a small head.
• Alopecia, epilepsy, pyorrhea, mental subnormality: A rare syndrome characterized by alopecia, epilepsy, mental retardation and pus-producing gum and tooth inflammations.
• Alopecia, mental retardation and neurological problems: A rare, newly described syndrome characterized by baldness, mental retardation and neurological problems.
• Alpers Syndrome: A rare syndrome characterized by liver disease, seizures and progressive, episodic psychomotor retardation.
• Alpha-Mannosidosis: A rare condition which is characterized by a lysosomal storage defect.
• Alpha-N-acetylgalactosaminidase deficiency, Type II: A very rare inherited metabolic disorder where deficiency of an enzyme (alpha-N-acetylgalactosaminidase) causes glycoplids to accumulate in body tissues and result in various symptoms. Type 2 occurs during the second or third decade of life and is milder than type I and doesn't involve neurological degeneration.
• Alpha-N-acetylgalactosaminidase deficiency, Type III: A very rare enzyme deficiency (N-acetyl-alpha-D-galactosaminidase) which can occur in three forms: type I (infantile-onset neuroaxonal dystrophy), type II or Kanzaki disease (adult-onset) and type III (mild or moderate form).
• Alpha-ketoglutarate dehydrogenase deficiency: A metabolic disorder characterized by a deficiency of Alpha-ketoglutarate dehydrogenase which results in high levels of oxoglutaric acid in the urine as well as other severe symptoms.
• Alpine syndrome: A condition that occurs in some people who go to low altitude winter resorts (1500 metres). It tends to mostly affect people who have been fasting when they arrive.
• Alternating Hemiplegia: Episodes of one-sided paralysis.
• Aluminium toxicity: High body levels of aluminium resulting in symptoms. Usually occurs in patients with renal impairment.
• Alveolar Hydatid Disease: Rare multi-organ tapeworm infection caught from animals.
• Alveolar echinococcosis: A rare parasitic infection caused by the larva of a miniscule tapeworm called Echinococcus multilocularis. Transmission occurs through contact with foxes, coyotes, dogs and cats. The condition results in the development of parasitic tumors in the liver (sometimes other organs such as brain and lungs) but it generally causes no symptom for 5 to 15 years after infection.
• Alzheimer disease 10: An inherited form of Alzheimer's. Type 10 is caused by a genetic defect on chromosome 10p13.
• Alzheimer disease 12: An inherited form of Alzheimer's. Type 12 is caused by a genetic defect on chromosome 8p12-q22.
• Alzheimer disease 13: An inherited form of Alzheimer's disease that is linked to a defect on chromosome 1q21. Alzheimer's disease is a progressive disorder involving degeneration of the brain. The disease mainly affects brain functions involving thinking, memory, personality and behaviour.
• Alzheimer disease 14: An inherited form of Alzheimer's disease that is linked to a defect on chromosome 1q25. Alzheimer's disease is a progressive disorder involving degeneration of the brain. The disease mainly affects brain functions involving thinking, memory, personality and behaviour.
• Alzheimer disease 15: An inherited form of Alzheimer's disease that is linked to a defect on chromosome 3q22-q24. Alzheimer's disease is a progressive disorder involving degeneration of the brain. The disease mainly affects brain functions involving thinking, memory, personality and behaviour.
• Alzheimer disease 16: Alzheimer disease 16 (late-onset) is a form of Alzheimer's disease that is linked to a defect on chromosome Xq21.3. Alzheimer's disease is a progressive disorder involving degeneration of the brain. The disease mainly affects brain functions involving thinking, memory, personality and behaviour.
• Alzheimer disease 2, late-onset: Alzheimer disease 2 (late-onset) is a form of Alzheimer's disease that is linked to a defect on chromosome 19q13.2. Alzheimer's disease is a progressive disorder involving degeneration of the brain. The disease mainly affects brain functions involving thinking, memory, personality and behaviour.
• Alzheimer disease 3, (early-onset Alzheimer disease): Alzheimer disease 3 is an early-onset form of Alzheimer's disease that is linked to a defect on chromosome 14q24.3. Alzheimer's disease is a progressive disorder involving degeneration of the brain. The disease mainly affects brain functions involving thinking, memory, personality and behaviour.
• Alzheimer disease 5: An inherited form of Alzheimer's. Type 5 has a late onset and is caused by a genetic defect on chromosome 12p11.
• Alzheimer disease 6: A genetic form of Alzheimer's. Type 6 has a late onset and is caused by a genetic defect on chromosome 10q24.
• Alzheimer disease 7: An inherited form of Alzheimer's. Type 7 is caused by a genetic defect on chromosome 10p13.
• Alzheimer disease 8: An inherited form of Alzheimer's. Type 8 is caused by a genetic defect on chromosome 20p.
• Alzheimer disease 9: A genetic form of Alzheimer's. Type 9 has a late onset and is caused by a genetic defect on chromosome 19p13.2.
• Alzheimer disease type 1: A degenerative brain disease characterized primarily by progressive dementia. Type 1 has an early onset (starts before the age of 65). It is caused by mutations in the APP gene which results in the production of a toxic protein (amyloid beta peptide) in the brain which collects into clumps (amyloid plaques) in the brain. These plaques cause damage to nerve cells in the brain.
• Alzheimer disease type 2: A degenerative brain disease characterized primarily by progressive dementia. Type 2 has a late onset - starts after the age of 65. It is believed to be caused by a combination of genetic mutations and environmental and lifestyle factors. The condition occurs when there is excessive production of a toxic protein (amyloid beta peptide) in the brain which collects into clumps (amyloid plaques) in the brain. These plaques cause damage to nerve cells in the brain.
• Alzheimer disease type 4: A degenerative brain disease characterized primarily by progressive dementia. Type 4 has an early onset (starts before the age of 65). It is caused by mutations in the PSEN2 gene which results in the production of a toxic protein (amyloid beta peptide) in the brain which collects into clumps (amyloid plaques) in the brain. These plaques cause damage to nerve cells in the brain.
• Alzheimer disease, early-onset, with cerebral amyloid angiopathy: An early-onset form of Alzheimer's disease that is linked to a defect on chromosome 21q21. Alzheimer's disease is a progressive disorder involving degeneration of the brain. The disease mainly affects brain functions involving thinking, memory, personality and behaviour.
• Alzheimer disease, familial: A degenerative brain disease characterized primarily by progressive dementia. The familial form is very rare and is completely inherited and has an early onset (usually in the 4th decade). It occurs when there is excessive production of a toxic protein (amyloid beta peptide) in the brain which collects into clumps (amyloid plaques) in the brain. These plaques cause damage to nerve cells in the brain.
• Alzheimer disease, familial, 1: An inherited form of Alzheimer's disease that is linked to a defect on chromosome 21q. Alzheimer's disease is a progressive disorder involving degeneration of the brain. The disease mainly affects brain functions involving thinking, memory, personality and behaviour.
• Alzheimer disease, familial, 11: An inherited form of Alzheimer's disease that is linked to a defect on chromosome 9p22.1. Alzheimer's disease is a progressive disorder involving degeneration of the brain. The disease mainly affects brain functions involving thinking, memory, personality and behaviour.
• Alzheimer disease, familial, 3, with spastic paraparesis and apraxia: This form of Alzheimer's is an early-onset form of Alzheimer's that is linked to a defect on chromosome 14q24.3. It is characterized by features which are atypical for Alzheimer's - spastic paraparesis which occurs before the dementia symptoms and apraxia. Alzheimer's disease is a progressive disorder involving degeneration of the brain. The disease mainly affects brain functions involving thinking, memory, personality and behaviour.
• Alzheimer disease, familial, 3, with spastic paraparesis and unusual plaques: This form of Alzheimer's is an early-onset form of Alzheimer's that is linked to a defect on chromosome 14q24.3. It is characterized by features which are atypical for Alzheimer's - spastic paraparesis which occurs before the dementia symptoms and unusual plaques in the brain. Alzheimer's disease is a progressive disorder involving degeneration of the brain. The disease mainly affects brain functions involving thinking, memory, personality and behaviour.
• Alzheimer disease, familial, 4: An inherited form of Alzheimer's disease that is linked to a defect on chromosome 1q31-q42. Alzheimer's disease is a progressive disorder involving degeneration of the brain. The disease mainly affects brain functions involving thinking, memory, personality and behaviour.
• Alzheimer disease, familial, type 3: A degenerative brain disease characterized primarily by progressive dementia. Type 3 has an early onset (starts before the age of 65). It is caused by mutations in the PSEN1 gene which results in the production of a toxic protein (amyloid beta peptide) in the brain which collects into clumps (amyloid plaques) in the brain. These plaques cause damage to nerve cells in the brain.
• Alzheimer's disease: A progressive degenerative disease of the brain of unknown cause
• Alzheimer's disease without Neurofibrillary tangles: A form of Alzheimer's that involves only plaques and no neurofibrillary tangles. This form tends to have an older age of onset and death and a shorter disease duration.
• Amaurosis congenita of Leber: A rare genetic eye disorder characterized by blindness at birth or within years as well as other eye abnormalities.
• Ambien overdose: Ambien is a prescription drug mainly used to treat insomnia. Excessive doses of the drug can result in various symptoms and even death in severe cases.
• Amelo-cerebro-hypohidrotic syndrome: A rare syndrome involving degeneration of the central nervous system, seizures and abnormal tooth development.
• American mountain fever: A viral disease transmitted through the bite of ticks (Rocky Mountain wood tick and American dog tick) who are infected with the virus. Because the virus infects blood cells including erythrocytes, transmission can also occur through transfusion with infected blood but this is uncommon. Infection is most common in Canada and parts of western US. The incubation period usually lasts between 3 and 6 days but can be as long as a few weeks. The virus tends to cause to periods of fever each lasting for a few days.
• Aminoacylase 1 deficiency: A rare genetic disorder caused by an enzyme (aminoacylase-1) deficiency. There is still uncertainty whether the deficiency actually causes any of the symptoms observed in patients.
• Amitriptyline toxicity: The toxic reaction of the body to the substance, possibly via allergic reaction or overdose.
• Amnesic shellfish poisoning: Rare shellfish poisoning sometimes causing amnesia.
• Amoxapine toxicity: The toxic reaction of the body to the substance, possibly via allergic reaction or overdose.
• Amphetamine abuse: Use of the stimulant drugs known as amphetamines or "speed"
• Amphetamine-induced hypertension: Amphetamine-induced hypertension is high blood pressure caused by use of amphetamines. Patients with an existing history of hypertension may suffer further blood pressure increases while taking amphetamines and this can be serious. Severity of symptoms varies amongst patients depending on their susceptibility, underlying health and duration of amphetamine use.
• Ampola syndrome: A rare genetic disease characterized primarily by mental retardation, facial anomalies, short stature, seizures and finger and toe abnormalities.
• Amyloid Neuropathies: A peripheral nerve disorder caused by abnormal amyloid deposits in the nerves. Sensory, autonomic or motor nerves may be affected. The degree of nerve involvement, and hence symptoms, are variable.
• Amyloid angiopathy: A blood vessel disorder caused by abnormal amyloid deposits in the blood vessel walls of the brain. The deposits can cause the blood vessel to become weak and rupture resulting in intracranial bleeding. Despite the potentially serious consequences the disorder is often asymptomatic until old age.
• Amyloid cardiopathy: Amyloidosis is a rare group of metabolic disorders where a protein called amyloid accumulates in body organs and tissues where it can cause damage and is potentially fatal. The cardiac form involves deposits of amyloid in the heart muscle which affects its function. The electrical conduction system of the heart is impaired.
• Amyloid cardiopathy, familial: Amyloidosis is a rare group of metabolic disorders where a protein called amyloid accumulates in body organs and tissues where it can cause damage and is potentially fatal. The familial cardiac form is inherited and involves deposits of amyloid in the heart muscle which affects its function. The electrical conduction system of the heart is impaired.
• Amyloidosis AL: A disease involving the abnormal deposit of amyloid fibrils in virtually any part of the body - the heart, liver, kidney and peripheral and autonomic nerves are most commonly affected. The abnormal amyloid fibrils are produced abnormal plasma cells in the bone marrow. In some cases, the excess growth of abnormal plasma cells can result in a cancerous condition called myeloma resulting in bone pain and infections. A patient with myeloma may develop amyloidosis but it is rare for a patient with AL amyloidosis to go on to develop myeloma.
• Amyloidosis VII: Amyloidosis involves the abnormal deposit of a substance called amyloid in various parts of the body. In the Ohio type, the amyloid deposits in the leptomeningeal blood vessels, brainstem, spinal cord and eye causing central nervous system dysfunction, brain hemorrhages as well as vision impairment.
• Amyloidosis beta2-microglobulinic: Amyloidosis is a rare group of metabolic disorders where a protein called amyloid accumulates in body organs and tissues where it can cause damage. The type of amyloid protein involved in this type of amyloidosis is beta-2-microglobulin. The abnormal protein tends to be deposited in parts of the body such as joints, bones and carpal tunnel but can also be found in the gastrointestinal tract and other organs.
• Amyloidosis, Familial: Amyloidosis is a rare group of metabolic disorders where a protein called amyloid accumulates in body organs and tissues where it can cause damage. In the familial form, the type of amyloid involved is usually a plasma protein called transthyretin. The main parts of the body affected are the nerves, heart and kidneys.
• Amyloidosis, Inherited: Amyloidosis is a rare group of metabolic disorders where a protein called amyloid accumulates in body organs and tissues where it can cause damage. In the inherited form, the type of amyloid involved is usually a plasma protein called transthyretin. The main parts of the body affected are the nerves, heart and kidneys.
• Amyloidosis, familial cutaneous: Amyloidosis involves the abnormal deposit of a substance called amyloid in various parts of the body. The familial cutaneous form is characterized by brown skin pigmentation as well as systemic symptoms such as failure to thrive, developmental delay, gastrointestinal problems and pneumonia.
• Amyloidosis, inflammatory: Amyloidosis is a rare group of metabolic disorders where a protein called amyloid accumulates in body organs and tissues where it can cause damage and is potentially fatal. Symptoms depend on the organs involved. Secondary amyloidosis is caused by a chronic infection of inflammatory conditions such as rheumatoid arthritis, multiple myeloma, tuberculosis and osteomyelitis. The main organs affected in secondary amyloidosis are usually the kidneys, liver, spleen and lymph nodes. The peripheral and autonomic nerves and the heart are rarely affected.
• Amyloidosis, oculoleptomeningeal: Amyloidosis involves the abnormal deposit of a substance called amyloid in various parts of the body. In this particular type, the amyloid deposits in the leptomeningeal blood vessels, brainstem, spinal cord and eye causing central nervous system dysfunction, brain hemorrhages and vision impairment.
• Amyoplasia: A rare condition characterized by congenital joint stiffness.
• Amyotrophic lateral sclerosis: A degenerative motor neuron disease marked by weakness and wasting of the muscles which starts at the hands and legs and spreads to the rest of the body. Death occurs in 2 to 5 years. Also called Lou Gehrig's disease or wasting palsy.
• Amyotrophic lateral sclerosis 2, juvenile: An inherited disorder involving progressive degeneration of motor neurons which results in muscle weakness and wasting. Type 2 is caused by a defect on chromosome 2q33.
• Amyotrophic lateral sclerosis 3: An inherited disorder involving progressive degeneration of motor neurons which results in muscle weakness and wasting. Type 3 is caused by a defect on chromosome 18q21.
• Amyotrophic lateral sclerosis 4, juvenile: An inherited disorder involving progressive degeneration of motor neurons which results in muscle weakness and wasting. Type 4 is caused by a defect on chromosome 9q34.
• Amyotrophic lateral sclerosis 5: An inherited disorder involving progressive degeneration of motor neurons which results in muscle weakness and wasting. Type 5 is caused by a defect on chromosome 15q15.1-q21.1.
• Amyotrophic lateral sclerosis 6: An inherited disorder involving progressive degeneration of motor neurons which results in muscle weakness and wasting. Type 6 is caused by a defect on chromosome 16q12.
• Amyotrophic lateral sclerosis 7: An inherited disorder involving progressive degeneration of motor neurons which results in muscle weakness and wasting. Type 7 is caused by a defect on chromosome 20p13.
• Amyotrophic lateral sclerosis 8: An inherited disorder involving progressive degeneration of motor neurons which results in muscle weakness and wasting. Type 9 is caused by a defect on chromosome 20q13.3 and is a dominantly inherited, late-onset form.
• Amyotrophic lateral sclerosis type 1:
• Amyotrophic lateral sclerosis, 11: An inherited progressive disease where destruction of motor nerves in the spinal cord and brain stem cause progressive muscle weakness and wasting. Type 11 is differentiated by the origin of the genetic defect involved (6q21).
• Amyotrophic lateral sclerosis, 9: An inherited progressive disease where destruction of motor nerves in the spinal cord and brain stem cause progressive muscle weakness and wasting. Type 9 is differentiated by the origin of the genetic defect involved (14q11).
• Amyotrophic lateral sclerosis, familial:
• Amyotrophic lateral sclerosis, familial type 1: A generally fatal, inherited progressive disease where destruction of motor nerves in the spinal cord and brain stem cause progressive muscle weakness and wasting. Type 1 is characterized by adult onset and relatively fast progression of symptoms. It usually occurs in an autosomal dominant pattern of inheritance.
• Amyotrophic lateral sclerosis, familial type 2: A generally fatal, inherited progressive disease where destruction of motor nerves in the spinal cord and brain stem cause progressive muscle weakness and wasting. Type 2 is characterized by childhood or adolescent onset of symptoms which progress very slowly over decades. It occurs in an autosomal recessive pattern of inheritance.
• Amyotrophic lateral sclerosis, familial type 3: A generally fatal, inherited progressive disease where destruction of motor nerves in the spinal cord and brain stem cause progressive muscle weakness and wasting. Type 3 is characterized late adulthood onset of symptoms which progress slowly over 5 years. It occurs in an autosomal dominant pattern of inheritance.
• Amyotrophic lateral sclerosis, familial type 4: A generally fatal progressive disease where destruction of motor nerves in the spinal cord and brain stem cause progressive muscle weakness and wasting. Type 4 is characterized by the onset of symptoms before the age of 25 and slow progression over the next few decades. It occurs in an autosomal dominant pattern of inheritance.
• Amyotrophic lateral sclerosis, familial type 5: A generally fatal, inherited progressive disease where destruction of motor nerves in the spinal cord and brain stem cause progressive muscle weakness and wasting. Type 6 is characterized adolescent onset of symptoms with progression varying between 1 and 20 years. It occurs in an autosomal recessive pattern of inheritance.
• Amyotrophic lateral sclerosis, familial type 6: A generally fatal, inherited progressive disease where destruction of motor nerves in the spinal cord and brain stem cause progressive muscle weakness and wasting. Type 6 is characterized adult onset of symptoms with progression varying between 1 and 20 years. It occurs in an autosomal dominant pattern of inheritance.
• Amyotrophic lateral sclerosis, familial type 7: A generally fatal, inherited progressive disease where destruction of motor nerves in the spinal cord and brain stem cause progressive muscle weakness and wasting. Type 7 is characterized adult onset of symptoms with progression varying between less than 5 years to several decades. It occurs in an autosomal dominant pattern of inheritance.
• Amyotrophic lateral sclerosis, familial type 8: A generally fatal, inherited progressive disease where destruction of motor nerves in the spinal cord and brain stem cause progressive muscle weakness and wasting. Type 8 is characterized by adult onset and relatively slow progression of symptoms. It occurs in an autosomal dominant pattern of inheritance.
• Amyotrophic lateral sclerosis, type 6: An inherited disorder involving progressive degeneration of motor neurons which results in muscle weakness and wasting. Type 6 is caused by a defect on chromosome 16q12.
• Amyotrophy, neurogenic scapuloperoneal, New England type: An inherited disorder involving muscle wasting and weakness in the shoulder and lower leg. The exact symptoms that occur may vary from patient to patient with males often being more affected than females. An interesting observation of this condition is that symptoms and rate of progression tends to be more severe with each passing generation.
• Anaemia, sideroblastic, X-linked -- ataxia: A very rare inherited disorder characterized by mild anemia and early onset neurological motor symptoms. The neurological symptoms tend to be relatively stable or slowly progressive with only occasional dependence on crutches or wheelchairs.
• Anchovy poisoning (clupeotoxin): Some anchovies contain toxins (Clupeotoxin) which can be poisonous to humans if eaten. Heat does not destroy the toxin and there is still uncertainty as to the origin of the toxin. The toxin appears to be present in higher concentrations in summer and is believed to be possible linked to the consumption of toxic food in its food web. The size and age of the anchovy does not appear to be related to the toxicity. The anchovies are found in coastal waters off Africa and the Caribbean, Indian and Pacific Oceans.
• Ancylostoma duodenale: An infestation with Ancylostoma duodenale which is a parasitic hookwork whichcan cause serious disease in humans - usually occurs in people who work barefoot in damp soil. The hookworms suck blood from the intestines of the host which can result in anemia if there is a large number of worms.
• Andersen disease: An rare inborn error of metabolism involving glycogen storage and characterized by cirrhosis and sometimes liver failure. Lack of the amyl-transglucosidase enzyme and abnormal glycogen causes the condition.
• Andrade's syndrome: An inherited condition characterized by deposits of an abnormal protein called amyloid in various parts of the body including organs. The condition mainly involves neurological symptoms.
• Anemia: Reduced red blood cells in the blood
• Anemia of pregnancy: Anemia of pregnancy is anemia that occurs during pregnancy. Women's bodies have a greater demand for iron during pregnancy and if intake is not sufficient, anemia can result. Anemia in pregnant women can lead to infant problems such as premature birth, fetal death, retarded growth and other problems.
• Anemia, Hemolytic, Warm Antibody: A rare autoimmune condition where the body's defense system attacks and destroys red blood cells. The onset of the condition is triggered by temperatures 37ºC or higher. The severity of the disorder is variable.
• Anemia, Iron-Deficiency: A lack of fully functioning red blood cells due to a deficiency of iron. The iron allows the body to make hemoglobin in red blood cells which in turn allows the red blood cell to carry oxygen.
• Anemia, Refractory, with Excess of Blasts: A bone marrow disease which results in insufficient red blood cells in the blood (anemia). The prognosis is poor with death usually occurring within a couple of years. There are two types: type 1 refers to cases where the level of blasts is less than 10% and type 2 refers to cases where the level of blasts is 10-20%. When too many immature blood cells (blasts) are produced by the bone marrow, the condition may progress to acute myeloid leukemia - occurs in about a quarter of cases in type 1 and a third of cases in type 2.
• Anemia, Refractory, with Excess of Blasts, type 1: A bone marrow disease which results in insufficient red blood cells in the blood (anemia). The prognosis is poor with death usually occurring within a couple of years. Type 1 refers to cases where the level of blasts is less than 10% and type 2 refers to cases where the level of blasts is 10-20%. When too many immature blood cells (blasts) are produced by the bone marrow, the condition may progress to acute myeloid leukemia - occurs in about a quarter of cases in type 1.
• Anemia, Refractory, with Excess of Blasts, type 2: A bone marrow disease which results in insufficient red blood cells in the blood (anemia). The prognosis is poor with death usually occurring within a couple of years. Type 1 refers to cases where the level of blasts is less than 10% and type 2 refers to cases where the level of blasts is 10-20%. When too many immature blood cells (blasts) are produced by the bone marrow, the condition may progress to acute myeloid leukemia - occurs in about a third of cases in type 2.
• Anemia, Sideroblastic: A rare blood disorder where abnormal utilization of iron results in the production of defective red blood cells which have excessive deposits of iron in them.
• Anemia, hypochromic microcytic: A blood disorder where red blood cells are too small and lack sufficient iron. It can be inherited or caused by insufficient iron in the diet or from a genetic disorder.
• Anemia, sideroblastic spinocerebellar ataxia: A rare inherited condition characterized by anemia at birth as well as spinocerebellar ataxia (impaired ability to control voluntary movements).
• Anemias, Sideroblastic: Sideroblastic anemias are a group of rare blood disorders where the bone marrow is unable to produce normal red blood cells. The body has enough iron but the red blood cells are unable to utilize it in a normal manner and anemia results. The red blood cells become overloaded with iron and are unable to carry out their normal functions. Some forms of sideroblastic anemia are inherited but most tend to be acquired due to such things as exposure to toxins and certain drugs, leukemia, inflammatory conditions such as rheumatoid arthritis and nutritional deficiencies (e.g. copper and pyridoxine deficiency). Inherited forms usually appear in childhood whereas acquired forms usually occur in adulthood.
• Anesthetic agent-induced liver damage: Damage or injury to the liver caused by exposure to anesthetic agents. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anesthetic agent-induced liver damage -- Chloroform: Damage or injury to the liver caused by exposure to an anesthetic agent called chloroform. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anesthetic agent-induced liver damage -- Cyclopropane: Damage or injury to the liver caused by exposure to an anesthetic agent called cyclopropane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anesthetic agent-induced liver damage -- Ether: Damage or injury to the liver caused by exposure to an anesthetic agent called ether. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anesthetic agent-induced liver damage -- Halothane: Damage or injury to the liver caused by exposure to an anesthetic agent called halothane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anesthetic agent-induced liver damage -- Methoxyflurane: Damage or injury to the liver caused by exposure to an anesthetic agent called methoxyflurane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anesthetic agent-induced liver damage -- Nitrous Oxide: Damage or injury to the liver caused by exposure to an anesthetic agent called nitrous oxide. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Aneurysm, intracranial berry: A bulge in a blood vessel in the brain. The bulge can rupture causing a stroke. They usually form as a result of high blood pressure and weak blood vessel walls in the brain. There are five different subtypes of intracranial berry aneurysms with each one caused by a defect in different gene. The defective gene increases and individuals risk for developing intracranial berry aneurysms.
• Aneurysm, intracranial berry, 1: A bulge in a blood vessel in the brain. The bulge can rupture causing a stroke. They usually form as a result of high blood pressure and weak blood vessel walls in the brain. There are five different subtypes of intracranial berry aneurysms with each one caused by a defect in different gene. The defective gene increases and individuals risk for developing intracranial berry aneurysms. Type 1 is caused by a defect on chromosome 7q11.2.
• Aneurysm, intracranial berry, 10: A bulge in a blood vessel in the brain. The bulge can rupture causing a stroke. They usually form as a result of high blood pressure and weak blood vessel walls in the brain. There are five different subtypes of intracranial berry aneurysms with each one caused by a defect in different gene. The defective gene increases and individuals risk for developing intracranial berry aneurysms. Type 10 is caused by a defect on chromosome 8q12.1.
• Aneurysm, intracranial berry, 2: A bulge in a blood vessel in the brain. The bulge can rupture causing a stroke. They usually form as a result of high blood pressure and weak blood vessel walls in the brain. There are five different subtypes of intracranial berry aneurysms with each one caused by a defect in different gene. The defective gene increases and individuals risk for developing intracranial berry aneurysms. Type 2 is caused by a defect on chromosome 19q13.
• Aneurysm, intracranial berry, 3: A bulge in a blood vessel in the brain. The bulge can rupture causing a stroke. They usually form as a result of high blood pressure and weak blood vessel walls in the brain. There are five different subtypes of intracranial berry aneurysms with each one caused by a defect in different gene. The defective gene increases and individuals risk for developing intracranial berry aneurysms. Type 3 is caused by a defect on chromosome 1p36.13-p34.3.
• Aneurysm, intracranial berry, 4: A bulge in a blood vessel in the brain. The bulge can rupture causing a stroke. They usually form as a result of high blood pressure and weak blood vessel walls in the brain. There are five different subtypes of intracranial berry aneurysms with each one caused by a defect in different gene. The defective gene increases and individuals risk for developing intracranial berry aneurysms. Type 4 is caused by a defect on chromosome 5p15.2-14.3.
• Aneurysm, intracranial berry, 5: A bulge in a blood vessel in the brain. The bulge can rupture causing a stroke. They usually form as a result of high blood pressure and weak blood vessel walls in the brain. There are five different subtypes of intracranial berry aneurysms with each one caused by a defect in different gene. The defective gene increases and individuals risk for developing intracranial berry aneurysms. Type 5 is caused by a defect on chromosome 2p13.
• Aneurysm, intracranial berry, 6: A bulge in a blood vessel in the brain. The bulge can rupture causing a stroke. They usually form as a result of high blood pressure and weak blood vessel walls in the brain. There are now six different subtypes of intracranial berry aneurysms with each one caused by a defect in different gene. The defective gene increases an individuals risk for developing intracranial berry aneurysms. Type 6 is caused by a defect on chromosome 9p21.
• Aneurysm, intracranial berry, 7: A bulge in a blood vessel in the brain. The bulge can rupture causing a stroke. They usually form as a result of high blood pressure and weak blood vessel walls in the brain. There are five different subtypes of intracranial berry aneurysms with each one caused by a defect in different gene. The defective gene increases and individuals risk for developing intracranial berry aneurysms. Type 7 is caused by a defect on chromosome 11q24-q25.
• Aneurysm, intracranial berry, 8: A bulge in a blood vessel in the brain. The bulge can rupture causing a stroke. They usually form as a result of high blood pressure and weak blood vessel walls in the brain. There are five different subtypes of intracranial berry aneurysms with each one caused by a defect in different gene. The defective gene increases and individuals risk for developing intracranial berry aneurysms. Type 8 is caused by a defect on chromosome 14q23.
• Aneurysm, intracranial berry, 9: A bulge in a blood vessel in the brain. The bulge can rupture causing a stroke. They usually form as a result of high blood pressure and weak blood vessel walls in the brain. There are five different subtypes of intracranial berry aneurysms with each one caused by a defect in different gene. The defective gene increases and individuals risk for developing intracranial berry aneurysms. Type 9 is caused by a defect on chromosome 2q33.1.
• Aneurysmal subarachnoid haemorrhage: Bleeding in the space around the brain that occurs from a leak in a weakened or dilated blood vessel under the arachnoid layer of the brain. Death can occur if treatment is not prompt.
• Angelman syndrome: A rare genetic disorder characterized by a puppet-like gait, fits of laughter and characteristic facial features.
• Angelman-Like Syndrome, X-linked: A very rare syndrome characterized mainly by mental retardation, mutism, facial anomalies, epilepsy and weak eye muscles. Males tended to have severe mental retardation whereas female carriers had mild or no mental retardation. Patients do eventually walk but then often lose this ability by the age of 10 years. Female carriers tend to have mild symptoms and males have severe symptoms - symptoms are variable to some degree.
• Angina: Angina is a particular type of pain related to heart conditions
• Angina pectoris: severe chest pain due to ischemia
• Angiokeratoma -- mental retardation -- coarse face: A rare inherited genetic syndrome characterized by mental retardation, coarse facial features and capillary hemangiomas.
• Angiomatosis, diffuse corticomeningeal, of Divry and Van Bogaert: A rare condition characterized by diffuse sclerosis and clusters of capillaries in parts of the brain as well as a marbled appearance to the skin.
• Angiostrongyliasis: Infection by a parasitic worm (Angiostrongylus). Infection can occur through eating contaminated raw animals such as snails, slugs, prawns or crabs which act as hosts to these parasites.
• Aniridia: A genetic disorder where part or all of the iris of one or both eyes is missing. The iris is the colored part of the eye. There are four forms of the disease: AN-1, AN-II, AN-III and AN-IV.
• Aniridia ataxia renal agenesis psychomotor retardation: A rare genetic disorder characterized by missing irises of the eye, ataxia, psychomotor retardation and abnormally kidneys.
• Aniridia cerebellar ataxia mental deficiency: A rare inherited disorder characterized by a partial absence of the iris, mental retardation and impaired coordination of voluntary movements.
• Ankle Osteoarthritis: A form of arthritis where the cartilage which cushions the bones in the ankle joint as they move against each other, becomes progressively degraded and damaged. This causes problems such as pain, stiffness and impaired movement. Pain is not present in all cases however, with some patients imply suffering joint stiffness. Primary osteoarthritis occurs as the person ages but results from repetitive use and/or high mechanical stress on the joint. It is not a direct result of the aging process. Secondary osteoarthritis is the result of such things as injury to the joint, joint infection, obesity, ligament damage, joint overuse, hormonal problems, pregnancy and various other conditions. Family history seem to play a factor in developing the condition.
• Ankle clonus: a rhythmic contraction of the calf muscles following a sudden dorsiflexion of the foot, the leg being semiflexed
• Ankle conditions: Conditions that affect the ankle
• Ankle fracture: Ankle fracture refers to a broken bone in the ankle.
• Ankle injuries: Injury to the ankle
• Ankle paralysis: A loss of the motor and or sensory function of the ankle due to either a muscular or neural mechanism
• Ankle spasm: A sudden involuntary contraction of the muscles surrounding the ankle joint.
• Ankle weakness: A decrease in the usual strength of the movements of the ankle
• Ankylosing Spondylitis: A form of chronic inflammation of the spine which may also affect joints in the shoulder, hip, neck, ribs and jaw. May result in loss of mobility. Also called Marie-Strumpell disease.
• Ankylosis: Stiffness or fusion of a joint leading to loss of mobility.
• Anophthalmia -- hypothalamo-pituitary insufficiency: A rare syndrome characterized mainly by small or absent eyes and malformations of the hypothalamus and pituitary gland.
• Anophthalmia -- hypyothalamo-pituitary insufficiency: A rare syndrome characterized mainly by small or absent eyes and malformations of the hypothalamus and pituitary gland.
• Anorexia Nervosa: A disorder where a distorted sense of body image leads to self-starvation to the point of death in some cases.
• Anorexia nervosa, genetic types: There is mounting evidence that anorexia nervosa may be caused by genetic factors which when combined with psychosocial factors can increase a persons risk of developing the condition.
• Anotia -- facial palsy -- cardiac defect: A rare syndrome characterized mainly missing ears, facial weakness and congenital heart defects.
• Anoxemia: Lack of oxygen in the blood.
• Anoxia: A condition marked by a lack of oxygen.
• Antalgic gait: Antalgic gait is a limping type of walk.
• Antalgic gait in adults: Antalgic gait in adults is an abnormal type of walk or limp in adults.
• Antalgic gait in children: Antalgic gait in children is an abnormal type of walk or limp in children.
• Antepartum Eclampsia: Antepartum eclampsia is the development of seizures or coma in pregnant women suffering from high blood pressure. Antepartum means that it occurs before delivery. Eclampsia is a serious condition which requires urgent medical treatment. Eclampsia may be associated with moderate as well as significant increases in blood pressure. The blood pressure can return to normal after delivery or may persist for a period of time.
• Anterior Interosseous Nerve Compression: Compression or entrapment of the radial nerve which is in the forearm. The problem can result from such things as bone tumors, trauma, lipomas or the repetition of certain arm motion. This nerve is involved in controlling various muscles in the hand and the wrist. The condition may be misdiagnosed as tennis elbow - the pain from tennis elbow is usually closer to the elbow than the pain in radial tunnel syndrome.
• Anterior knee pain related to sports: The knee joint is the largest joint in the body, consisting of 4 bones and an extensive network of ligaments and muscles. Injuries to the knee joint are amongst the most common in sporting activities.
• Anterior pituitary hyperhormonotrophic syndrome: A syndrome characterized by the excessive production of various hormones (gonadotrophic, thyrotrophic, lactotrophic and pancreatrophic hormone).
• Anterior spinal artery stroke: An interruption to the blood supply in the anterior spinal artery which affects sensation, motor control and bowel control. The symptoms may improve to varying degrees once the blood supply returns to normal. The severity of the disorder depends on the exact location of the defect and how long it persists for.
• Anterior spinal artery syndrome: Neurological symptoms caused by the blockage of the anterior spinal artery. The blockage may be caused by such things as trauma, cancer, thrombosis and arterial disease. Symptoms are determined by the exact location of the blockage.
• Antibiotics-induced liver damage: Damage or injury to the liver caused by taking certain antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage -- Cephalosporin: Damage or injury to the liver caused by exposure to Cephalosporin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage -- Chloramphenicol: Damage or injury to the liver caused by exposure to an antibiotic called Chloramphenicol. Chloramphenicol a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage -- Clindamycin: Damage or injury to the liver caused by exposure to Clindamycin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage -- Erythromycin Ethyl succinate: Damage or injury to the liver caused by exposure to Erythromycin Ethyl succinate antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage -- Erythromycin estolate: Damage or injury to the liver caused by exposure Erythromycin estolate antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage -- Nitrofuran: Damage or injury to the liver caused by exposure to Nitrofuran antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage -- Novobiocin: Damage or injury to the liver caused by exposure to Novobiocin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage -- Penicillin: Damage or injury to the liver caused by exposure to Penicillin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage -- Quinolone: Damage or injury to the liver caused by exposure to Quinolone antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage -- Rifampicin: Damage or injury to the liver caused by exposure to Rifampicin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage -- Spectinomycin: Damage or injury to the liver caused by exposure to Spectinomycin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage -- Sulfones: Damage or injury to the liver caused by exposure to sulfone antiboitics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage -- Telithromycin: Damage or injury to the liver caused by exposure to Telithromycin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage -- Tetracycline: Damage or injury to the liver caused by exposure to Tetracycline antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anticonvulsive-induced liver damage: Damage or injury to the liver caused by exposure to anticonvulsives. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anticonvulsive-induced liver damage -- Mephenytoin: Damage or injury to the liver caused by exposure to an anticonvulsive called mephenytoin. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anticonvulsive-induced liver damage -- Phenobarbital: Damage or injury to the liver caused by exposure to an anticonvulsive called phenobarbital. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anticonvulsive-induced liver damage -- Phenytoin: Damage or injury to the liver caused by exposure to an anticonvulsive called Phenytoin. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anticonvulsive-induced liver damage -- Valproic Acid: Damage or injury to the liver caused by exposure to an anticonvulsive called valproic acid. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antifungal agent-induced liver damage: Damage or injury to the liver caused by exposure to antifungal agents. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antifungal agent-induced liver damage -- 5-Fluorocytosine: Damage or injury to the liver caused by exposure to an antifungal agent called 5-Fluorocytosine. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antifungal agent-induced liver damage -- Amphotericin: Damage or injury to the liver caused by exposure to an antifungal agent called Amphotericin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antifungal agent-induced liver damage -- Griseofulvin: Damage or injury to the liver caused by exposure to an antifungal agent called Griseofulvin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antifungal agent-induced liver damage -- Ketoconazole: Damage or injury to the liver caused by exposure to an antifungal agent called Ketoconazole. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antifungal agent-induced liver damage -- Saramycetin: Damage or injury to the liver caused by exposure to an antifungal agent called Saramycetin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antimetazoal agent-induced liver damage: Damage or injury to the liver caused by exposure to Antimetazoal agents. Antimetazoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antimetazoal agent-induced liver damage -- Amodiaquine: Damage or injury to the liver caused by exposure to an antimetazoal agent called amodiaquine. Antimetazoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antimetazoal agent-induced liver damage -- Hycanthone: Damage or injury to the liver caused by exposure to an antiprotozoal agent called hycanthone. Antimetazoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage: Damage or injury to the liver caused by exposure to antiprotozoal agents. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage -- 8-Hydroxyquinolone: Damage or injury to the liver caused by exposure to an antiprotozoal agent called 8-Hydroxyquinolone. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage -- Carbarsone: Damage or injury to the liver caused by exposure to an antiprotozoal agent called carbarsone. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage -- Emetine: Damage or injury to the liver caused by exposure to an antiprotozoal agent called emetine. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage -- Mepacrine: Damage or injury to the liver caused by exposure to an antiprotozoal agent called mepacrine. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage -- Metronidazole: Damage or injury to the liver caused by exposure to an antiprotozoal agent called Metronidazole. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage -- Thiabendazole: Damage or injury to the liver caused by exposure to an antiprotozoal agent called Thiabendazole. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antituberculous agent-induced liver damage: Damage or injury to the liver caused by exposure to antituberculous agents. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antituberculous agent-induced liver damage -- Cycloserine: Damage or injury to the liver caused by exposure to an antituberculous agent called cycloserine. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antituberculous agent-induced liver damage -- Ethionamide: Damage or injury to the liver caused by exposure to an antituberculous agent called ethionamide. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antituberculous agent-induced liver damage -- Isoniazid: Damage or injury to the liver caused by exposure to an antituberculous agent called isoniazid. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antituberculous agent-induced liver damage -- Rifampicin: Damage or injury to the liver caused by exposure to an antituberculous agent called rifampicin. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antituberculous agent-induced liver damage -- p-aminosalicylic acid: Damage or injury to the liver caused by exposure to an antituberculous agent called p-aminosalicylic acid. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiviral agent-induced liver damage: Damage or injury to the liver caused by exposure to antiviral agents. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiviral agent-induced liver damage -- Cytarabine: Damage or injury to the liver caused by exposure to an antiviral agent called cytarabine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiviral agent-induced liver damage -- Vidarabine: Damage or injury to the liver caused by exposure to an antiviral agent called vidarabine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiviral agent-induced liver damage -- idoxuridine: Damage or injury to the liver caused by exposure to an antiviral agent called idoxuridine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiviral agent-induced liver damage -- xenylamine: Damage or injury to the liver caused by exposure to an antiviral agent called xenylamine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antley-Bixler Syndrome: A rare genetic disorder characterized by premature closing of skull bones, choanal atresia and craniofacial and limb abnormalities.
• Antley-Bixler-like syndrome -- ambiguous genitalia -- disordered steroidogenesis: A rare genetic disorder involving a deficiency of an enzyme (cytochrome P450 oxidoreductase) which causes steroid abnormalities. The condition results in ambiguous genitalia in females due to excessive androgen during fetal growth. Patients can also have the bone symptoms of Antley-Bixler syndrome.
• Anxiety: Excessive worry, anxiety, or fear.
• Anxiety disorder: A mental condition that is characterized by anxiety and avoidance behaviours
• Anxiety-tension syndrome: Anxiety associated with physical symptoms such as tense muscles and fatigue.
• Aorta-pulmonary artery fistula: An abnormal opening or connection between the aorta and the main pulmonary artery. It can occur through a traumatic penetrating injury or may be a complication of surgery. Severe cases can lead to heart failure.
• Aortic Valve Insufficiency: A heart valve disorder where the heart valve is unable to close completely which causes a backflow of some of the blood from the aorta. The condition can be caused by such things as systemic lupus erythematosus, endocarditis, high blood pressure, Marfan's syndrome and aortic dissection.
• Aortic arches defect: A defect in the top part of the aorta (aortic arch) that consists of several arterial branches. There is a variety of defects that can occur and symptoms will be determined by the particular defect involved. Possible types of defects includes aortic coarctation and aortic arch hypoplasia.
• Aortic coarctation: A rare inherited birth defect where the heart blood vessel called the aorta has a narrowed area which affects blood flow. The degree of constriction is variable which mild cases asymptomatic until adulthood. The poor blood flow to the lower body gives results in it appearing less developed than that upper body.
• Aortic dilatation- joint hypermobility- arterial tortuosity: A rare syndrome characterized by the dilation of the aortic root, hyperextensible joints and varicose veins
• Aortic stenosis: A condition which affects the aortic valve of the heart resulting in stenosis of the valve.
• Aortic valve disease: Disease of the heart's aortic valve
• Aortic valve incompetence: A condition characterized by an inability of the aortic valve to function effectively
• Aortic valve stenosis: A congenital condition involving a malformation of the valve that controls the blood flow of the main heart vessel (aorta). The valve doesn't open enough to allow sufficient blood to flow through the aorta which reduces the supply of oxygenated blood to the body.
• Aortic valves stenosis of the child: A birth defect where the aortic valve is abnormally narrow or unable to fully open. Depending on the degree of narrowing, the symptoms may range from severe to asymptomatic.
• Apallic syndrome: A persistent vegetative state caused by brain damage.
• Aphasia-epilepsy, acquired: A rare childhood neurological disorder characterized by aphasia, epileptic seizures and inability to recognize sounds. The symptoms may develop quickly or gradually.
• Aplastic anemia: A blood disorder where the bone marrow produces insufficient new blood cells.
• Apo A-I deficiency: Low plasma HDL cholesterol that tends to run in families.
• Appian-Plutarch syndrome: Symptoms caused by excessive doses of a drug called atropine.
• Apple seed poisoning: Apple seeds contain a toxic chemical called amygdalin which can cause serious symptoms if eaten in large quantities. Hospital admission is recommended if more than 50 apple seeds have been consumed.
• Apraxia: A disorder of skilled movement not due to tremors, weakness, akinesia or abnormal tone or posture.
• Apraxia in children: Apraxia in children refers to a child who has difficulty in consistently and correctly saying what is intended as the result of a disorder of the central nervous system.
• Apricot seed poisoning: Apricot seeds contain a chemical called amygdalin which breaks down into cyanide in the human body. The toxic chemicals are not released if the pit remains intact and therefore poisoning usually occurs if the seeds are crushed and eaten. Accidental ingestion is very unusual. Most parts of the apricot plant contain the toxic chemical with the highest concentration in young leaves. Different species of apricots have different levels of toxic chemical. Severe symptoms or even death can occur if children consume more than ten kernels or adults consume more than forty kernels. Theories exist that apricot kernels may help cancer sufferers but there has been no scientific studies that have proven this.
• Arachnodactyly -- ataxia -- cataract -- aminoaciduria -- mental retardation: A rare syndrome characterized mainly by congenital cataracts, ataxia, mental retardation, abnormal amino acid metabolism and long, thin fingers.
• Arachnoid Cysts: A rare disorder involving a fluid-filled cysts on the arachnoid membrane which is one of the thin layers of tissue that form a membrane which covers the spinal cord and brain. The type and severity of symptoms is determined by the size and location of the cyst.
• Arachnoiditis: A progressive disorder where the arachnoid membrane becomes inflamed and the brain and spinal cord may also become inflamed.
• Arakawa's syndrome 2: An inherited metabolic disorder where an enzyme deficiency (methionine synthase) causes mental and physical retardation, blood disorders, degeneration of brain tissue and various other symptoms.
• Arbovirosis: An infectious disease caused by an arbovirus. The virus is transmitted by arthropods such as insects and ticks. Examples of arboviruses include Yellow Fever, Japanese encephalitis and tick-borne encephalitis. The symptoms may vary depending on the type of virus involved. The infection can lead to life-threatening brain inflammation.
• Arctic bearded seal poisoning: The Arctic Bearded seal is often used as a food source by the arctic inhabitants. Eating the liver and kidneys of the arctic bearded seal can result in a Vitamin A overdose which can cause serious symptoms and even death in extreme cases. It is believed that eating more than 100-250 grams of the seal liver can result in human death.

Conditions listing medical symptoms: Movement symptoms:

The following list of conditions have 'Movement symptoms' or similar listed as a symptom in our database. This computer-generated list may be inaccurate or incomplete. Always seek prompt professional medical advice about the cause of any symptom.

Select from the following alphabetical view of conditions which include a symptom of Movement symptoms or choose View All.

Conditions listing medical complications: Movement symptoms:

The following list of medical conditions have 'Movement symptoms' or similar listed as a medical complication in our database.

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