Glossary for Spleen symptoms

Medical terms related to Spleen symptoms or mentioned in this section include:

4-Alpha-hydroxyphenylpyruvate hydroxylase deficiency: A very rare metabolic disorder where a deficiency of a particular enzyme results in the urinary excretion of a chemical called hawkinsin. Symptoms start once the infant is weaned off breast milk.
ALL-Down syndrome: The presence of acute lymphoblastic leukemia in Down syndrome patients. These patients tend to have a poorer prognosis for the leukemia than patients without Down syndrome.
AREDYLD: A rare condition characterized by abnormalities of the extremities, teeth, hair, nail and kidney as well as lipoatrophic diabetes.
Aagenaes syndrome: A rare inherited birth abnormality involving underdeveloped lymph vessels which results in swollen legs and liver problems.
Abdominal symptoms: Symptoms affecting the abdomen or digestive tract
Acanthocytosis: A rare disorder where most of the red blood cells are abnormal with spiny projections due to lipid abnormalities. The blood abnormality is seen in conditions such as abetalipoproteinemia, severe liver disease and severe malnutrition. Symptoms and prognosis depend on the underlying disorder.
Achrestic anemia: Achrestic anemia is a form of anemia similar to that caused by Vitamin B12 deficiency but it doesn't respond to treatment with Vitamin B12. The condition tends to progress slowly and can result in death if not treated. There are a variety of possible causes.
Acidemia, methylmalonic: An inborn error of metabolism where amino acids in the body aren't metabolized properly resulting in high levels of the acid throughout the body.
Acquired idiopathic sideroblastic anaemia: A rare disorder where iron is transported into a developing blood cells but because it is unable to be used, it builds up within the cell and tends to stop it from developing into a fully functioning red blood cell. Thus anemia can occur despite adequate or even high iron levels. Acquired cases can occur on exposure to excess alcohol, lead and drugs or can occur to nutritional problems involving a deficiency of folic acid or copper or an excess of zinc. The condition can also be caused by conditions such as kidney problems, endocrine dysfunction, metabolic disorders, rheumatoid arthritis and leukemia.
Acquired total lipodystrophy: A rare acquired disorder that involves adipose tissue abnormalities and is characterized by loss of adipose tissue through the body. The disorder is very similar to the congenital form but the liver involvement is more severe.
Acral dysostosis -- dyserythropoiesis: A rare disorder characterized by hand and foot defects as well as a congenital form of anemia characterized by the production of abnormal red blood cells.
Acromegaly: An abnormal enlargement of the limbs due to increased secretion of growth hormone after the cessation of puberty
Actinomycetales infection: A bacterial infection from the order of Actinobacteria. The range of symptoms is variable depending on which bacteria from the order is involved.
Acute adult T-Cell leukemia: A form of blood cancer affecting the T-cells which make up the body's immune system. The disease is caused by the HTLV-1 virus (human T-cell leukemia virus) which causes the proliferation of abnormal T-cells. The virus can be transmitted sexually and may lay dormant for decades. There are four subtypes: acute, chronic, lymphoma and smoldering. The acute and lymphoma subtypes have the poorest prognosis. The acute subtype tends to progress rapidly and is the most prevalent form of the condition.
Acute basophilic leukaemia: A rare type of acute myeloid leukemia characterized by the presence of abnormal basophils.
Acute biphenotypic leukemia: A rare form of leukemia that has myeloid and lymphoid features.
Acute cholinergic dysautonomia: A rare condition characterized by the presence of abnormal red blood cells in the bone marrow and blood. The condition is characterized by anemia and generally leads to the development of acute myelogenous leukemia. The acute form has more severe symptoms than the chronic form.
Acute leukaemia of ambiguous lineage: A term used to describe a type of leukemia (a blood cancer) where the leukemic cells cannot be determined as myeloid or lymphoid or where both types of cells are present.
Acute leukemia: An acute condition which affects a cell line of the blood which shows little or no differentiation
Acute lymphoblastic leukemia: A malignant disease that starts suddenly and progresses quickly. It is characterized by a high number of immature cells in the organs, bone marrow and blood. Symptoms include fever, pallor, anorexia, fatigue, anemia, hemorrhage, bone pain, splenomegaly and frequent infections. Also called acute lymphocytic leukemia.
Acute lymphoblastic leukemia, Susceptibility to: Cancer of the white blood cells characterized by the presence of excessive lymphoblasts. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets. There are two subtypes of leukemia linked to a genetic anomaly which increases a person's susceptibility to developing the cancer. Type 1 is linked to a defect on chromosome 10q21 and type 2 is linked to a defect on chromosome 7p12.2.
Acute lymphoblastic leukemia, Susceptibility to, 1: Cancer of the white blood cells characterized by the presence of excessive lymphoblasts. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets. There are two subtypes of leukemia linked to a genetic anomaly which increases a person's susceptibility to developing the cancer. Type 1 is linked to a defect on chromosome 10q21.
Acute lymphoblastic leukemia, Susceptibility to, 2: Cancer of the white blood cells characterized by the presence of excessive lymphoblasts. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets. There are two subtypes of leukemia linked to a genetic anomaly which increases a person's susceptibility to developing the cancer. Type 2 is linked to a defect on chromosome 7p12.2.
Acute lymphoblastic leukemia, adult: Cancer of the white blood cells. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets.
Acute megacaryoblastic leukemia: A rare form of malignant bone marrow cancer involving the proliferation of immature precursors of blood cells. More specifically, it involves the rapid proliferation of megakaryoblasts (premature form of megakaryocytes).
Acute myeloblastic leukemia type 1: A form of blood cancer resulting in the rapid proliferation of immature blood cells (blast cells).
Acute myeloblastic leukemia type 2: A form of blood cancer resulting in the rapid proliferation of granulocytes and monocytes.
Acute myeloblastic leukemia type 3: A rare form of malignant bone marrow cancer involving the rapid proliferation of immature precursors of blood cells. Type 3 involves the proliferation of promyelocytes.
Acute myeloblastic leukemia type 4: A rare form of malignant bone marrow cancer involving the rapid proliferation of immature precursors of blood cells. Type 4 involves the rapid proliferation of myelocytes and monocytes.
Acute myeloblastic leukemia type 5: A rare form of malignant bone marrow cancer involving the proliferation of immature precursors of blood cells. Type 5 involves the rapid proliferation of monoblasts (immature precursors of monocytes) in particular.
Acute myeloblastic leukemia type 6: A rare form of malignant bone marrow cancer involving the rapid proliferation of immature precursors of blood cells. Type 6 involves the proliferation of the immature precursors of red blood cells called erythroblasts.
Acute myeloblastic leukemia type 7: A rare form of malignant bone marrow cancer involving the proliferation of immature precursors of blood cells. Type 7 involves the rapid proliferation of megakaryoblasts (premature form of megakaryocytes) in particular.
Acute myelocytic leukemia: A malignant cancer of blood-forming tissues resulting in a high number of immature leukocytes. Symptoms include soft bleeding gums, anemia, fatigue, fever, dyspnea, moderate splenomegaly, joint and bone pains and frequent infections. Also called acute granulocytic leukemia, acute myelogenous leukemia, acute nonlymphocytic leukemia, myeloid leukemia, splenomedullary leukemia, splenomyelogenous leukemia.
Acute myelofibrosis: A rare condition where progressive scarring or fibrosis of the bone marrow impairs it's ability to make blood cells causing symptoms such as anemia and liver and spleen enlargement.
Acute myeloid leukaemia and myelodysplastic syndromes related to alkylating agent: The use of alkylating agents to treat cancer can result in leukemia in some patients.
Acute myeloid leukaemia and myelodysplastic syndromes related to topoisomerase type II inhibitor: The use of topoisomerase type II inhibitors to treat cancer can result in leukemia in some patients.
Acute myeloid leukaemia and myelodysplastic syndromes, therapy related: Certain cancer therapies can result in the development of leukemia in some patients. These therapies includes topoisomerase type II inhibitors and alkylating agents.
Acute myeloid leukemia: A form of rapidly progressing blood cancer resulting in the rapid proliferation of granulocytes and monocytes, red blood cells and platelets.
Acute myeloid leukemia, adult: A form of blood cancer resulting in the rapid proliferation of granulocytes and monocytes, red blood cells and platelets.
Acute myelosclerosis: A rare disorder where the bone marrow makes too many blood cells. The disease progresses rapidly with death usually occurring within 6 months of onset.
Acute non lymphoblastic leukemia: A form of rapidly progressing blood cancer resulting in the rapid proliferation of granulocytes and monocytes, red blood cells and platelets. It is one of the most common forms of leukemia in adults but can occur in children.
Acute panmyelosis: A rare condition where progressive scarring or fibrosis of the bone marrow impairs it's ability to make blood cells causing symptoms such as anemia and liver and spleen enlargement. The disease often progresses rapidly and results in death.
Acute vitamin A toxicity: Acute ingestion of vitamin A can cause symptoms. Symptoms usually only last for a day or two.
Acyl-CoA dehydrogenase, very long chain, deficiency of: A rare inherited genetic condition where the body is unable to convert certain fats to energy i.e. there is not enough of a certain enzyme which is needed to metabolize a type of fat called long-chain fatty acids. The build-up of these fatty acids in the body causes damage. There are three subtypes of the disorder each with varying severity: severe early-onset form, an intermediate form and an adult-onset form.
Addison-Gull syndrome: A condition where scarring of the liver and bile ducts which results in chronic jaundice, enlarged spleen and liver and yellow skin plaques due to abnormal lipid metabolism.
Adrenoleukodystrophy: A rare hereditary metabolic disease that only occurs in male children and is characterized by adrenal atrophy and extensive cerebral demyelination causing progressive loss of mental functioning, aphasia, apraxia and sometimes blindness. The patient usually dies within 5 years.
Adrenoleukodystrophy, autosomal, neonatal form: A rare inherited disorder involving the adrenal glands, testes and certain parts of the brain (white matter). It is a less severe form of leukodystrophy where an abnormality within the body cells prevents the metabolism of certain fats (long chain fatty acids).
Adult T-Cell leukemia: A form of blood cancer affecting the T-cells which make up the body's immune system. The disease is caused by the HTLV-1 virus (human T-cell leukemia virus) which causes the proliferation of abnormal T-cells. The virus can be transmitted sexually and may lay dormant for decades. There are four subtypes: acute, chronic, lymphoma and smoldering. The acute and lymphoma subtypes have the poorest prognosis.
Adult T-Cell lymphoma: A form of blood cancer affecting the T-cells which make up the body's immune system. The disease is caused by the HTLV-1 virus (human T-cell leukemia virus) which causes the proliferation of abnormal T-cells. The virus can be transmitted sexually and may lay dormant for decades. There are four subtypes: acute, chronic, lymphoma and smoldering. The acute and lymphoma subtypes have the poorest prognosis. The lymphoma subtype is aggressive and tends to affect the lymph nodes more than the blood.
Adult onset Still's disease: A form of Still's disease that has a later onset and involves arthralgia or arthritis and a characteristic rash that often appears during periods of temperature increase.
Aflatoxicosis: Poisoning from ingestion of aflatoxins.
Aflatoxin B1 exposure: Aflatoxin B1 is a toxin produced by fungus from the Aspergillus genus. The toxin is carcinogenic in humans and can also cause other health effect, particularly liver problems. The fungus are found frequently in nature and can readily contaminate crops before harvest or during storage. It is common in moist soils and decaying vegetation. There are more than 13 different subtypes of aflatoxin with B1 being the most toxic. The condition is most common in poorly developed countries where there are insufficient controls on the presence of aflatoxin in food.
Aflatoxin B2 exposure: Aflatoxin B2 is a toxin produced by fungus from the Aspergillus genus. The toxin is carcinogenic in humans and can also cause other health effect, particularly liver problems. The fungus are found frequently in nature and can readily contaminate crops before harvest or during storage. It is common in moist soils and decaying vegetation. There are more than 13 different subtypes of aflatoxin with B1 being the most toxic. The condition is most common in poorly developed countries where there are insufficient controls on the presence of aflatoxin in food.
Aflatoxin G1 exposure: Aflatoxin G1 is a toxin produced by fungus from the Aspergillus genus. The toxin is carcinogenic in humans and can also cause other health effect, particularly liver problems. The fungus are found frequently in nature and can readily contaminate crops before harvest or during storage. It is common in moist soils and decaying vegetation. There are more than 13 different subtypes of aflatoxin with B1 being the most toxic. The condition is most common in poorly developed countries where there are insufficient controls on the presence of aflatoxin in food.
Aflatoxin G2 exposure: Aflatoxin G2 is a toxin produced by fungus from the Aspergillus genus. The toxin is carcinogenic in humans and can also cause other health effect, particularly liver problems. The fungus are found frequently in nature and can readily contaminate crops before harvest or during storage. It is common in moist soils and decaying vegetation. There are more than 13 different subtypes of aflatoxin with B1 being the most toxic. The condition is most common in poorly developed countries where there are insufficient controls on the presence of aflatoxin in food.
Aflatoxin exposure: Aflatoxins are toxins produced by fungus from the Aspergillus genus. The toxin is carcinogenic in humans and can also cause other health effect, particularly liver problems. The fungus are found frequently in nature and can readily contaminate crops before harvest or during storage. It is common in moist soils and decaying vegetation. There are more than 13 different subtypes of aflatoxin with B1 being the most toxic. The condition is most common in poorly developed countries where there are insufficient controls on the presence of aflatoxin in food.
African Sleeping sickness: A disease caused by parasites (Trypanosome brucei gamiense or T. brucei rodesiense) and transmitted to humans by the tsetse fly which is found only in Africa. Causes symptoms such as fever, chills, headache, anemia, edema of hands and feet, enlarged lymph glands, lethargy, sleepiness, convulsions and coma. Also called African trypanosomiasis and sleeping sickness.
Aggressive NK-cell leukaemia: An aggressive form of blood cancer involving the rapid proliferation of natural killer (NK) cells.
Aggressive systemic mastocytosis: The excessive proliferation of mast cells. Mast cells control the skin's response to minor injury and release a chemical called histamine which causes the skin to redden. In the aggressive form, mast cells accumulate in the liver, spleen and lymphatic system.
Aicardi-Goutieres syndrome 1: A rare inherited progressive disease that affects the brain and immune system. Type 1 is caused by a defect on chromosome 3p21.3-p21.2.
Aicardi-Goutieres syndrome 2: A rare inherited progressive disease that affects the brain and immune system. Type 2 is caused by a defect on chromosome 13q14-q21.
Aicardi-Goutieres syndrome 3: A rare inherited progressive disease that affects the brain and immune system. Type 3 is caused by a defect on chromosome 11q13.2.
Aicardi-Goutieres syndrome 4: A rare inherited progressive disease that affects the brain and immune system. Type 4 is caused by a defect on chromosome 19p13.13.
Aicardi-Goutieres syndrome 5: A rare inherited progressive disease that affects the brain and immune system. Type 5 is caused by a defect on chromosome 3p21.3-p21.2.
Alagille syndrome: A genetic disorder affecting the liver and characterized by the absence of some or all of the liver bile ducts that transport bile within the liver.
Albers-Schonberg disease -- malignant recessive form: A rare disorder characterized primarily by increased bone density as old bone is not resorbed and replaced with new bone - is also known as marble bone disease. The malignant infantile form is the most severe form of this disorder and death usually occurs in the first decade of life.
Alcohol-induced pseudo-Cushing syndrome: The excessive consumption of alcohol can result in symptoms similar to a condition called Cushing's syndrome. When alcohol consumption is stopped, symptoms regress.
Aldolase A deficiency: A rare condition where a deficiency of the enzyme called aldolase A causes muscle problems and anemia.
Alpha thalassemia: Thalassemia is an inherited blood disorder characterized by abnormal synthesis of hemoglobin. Hemoglobin consists of two main protein chains called alpha and beta. Alpha thalassemia involves defects in one or more of the four genes required to make each ? protein chain. The main symptom is anemia, the severity of which can vary amongst patients depending on how many defective genes are involved.
Alpha thalassemia -- Hemoglobin H disease: Thalassemia is an inherited blood disorder characterized by abnormal synthesis of hemoglobin. Hemoglobin consists of two main protein chains called alpha and beta. Hemoglobin H disease involves defects in three of the four genes required to make each ? protein chain. The main symptom is moderate to severe anemia.
Alpha thalassemia major: Thalassemia is an inherited blood disorder characterized by abnormal synthesis of hemoglobin. Hemoglobin consists of two main protein chains called alpha and beta. Alpha thalassemia major is very rare involves defects in all of the four genes required to make each ? protein chain. The condition leads to infant death before or soon after birth.
Alpha-Mannosidosis: A rare condition which is characterized by a lysosomal storage defect.
Alpha-mannosidosis type II: A rare inherited metabolic disorder involving a deficiency of an enzyme (alpha-mannosidosase) which results in the accumulation of certain chemicals in the body which leads to progressive damage. This form of the condition is less severe than type I (infantile form).
Alpha-mannosidosis, adult-onset form:
Amyloidosis: A disease characterized by the accumulation of insoluble amyloid protein in tissues and organs which in turn affects the functioning of these tissues and organs.
Amyloidosis AL: A disease involving the abnormal deposit of amyloid fibrils in virtually any part of the body - the heart, liver, kidney and peripheral and autonomic nerves are most commonly affected. The abnormal amyloid fibrils are produced abnormal plasma cells in the bone marrow. In some cases, the excess growth of abnormal plasma cells can result in a cancerous condition called myeloma resulting in bone pain and infections. A patient with myeloma may develop amyloidosis but it is rare for a patient with AL amyloidosis to go on to develop myeloma.
Amyloidosis, familial visceral: A rare genetic disorder involving widespread amyloidosis (abnormal buildup of amyloid protein in tissues) which tends the affect the kidneys severely.
Amyloidosis, inflammatory: Amyloidosis is a rare group of metabolic disorders where a protein called amyloid accumulates in body organs and tissues where it can cause damage and is potentially fatal. Symptoms depend on the organs involved. Secondary amyloidosis is caused by a chronic infection of inflammatory conditions such as rheumatoid arthritis, multiple myeloma, tuberculosis and osteomyelitis. The main organs affected in secondary amyloidosis are usually the kidneys, liver, spleen and lymph nodes. The peripheral and autonomic nerves and the heart are rarely affected.
Andersen disease: An rare inborn error of metabolism involving glycogen storage and characterized by cirrhosis and sometimes liver failure. Lack of the amyl-transglucosidase enzyme and abnormal glycogen causes the condition.
Anemia: Reduced red blood cells in the blood
Anemia, Blackfan Diamond: Diamond-Blackfan anemia is a rare genetic condition where the bone marrow is unable to make sufficient red blood cells which leads low levels of red blood cells. There are eight subtypes of the condition which differ in the location of the genetic defect and the incidence of additional symptoms such as malformations. The severity of symptoms is variable but most cases are serious.
Anemia, Hemolytic, Warm Antibody: A rare autoimmune condition where the body's defense system attacks and destroys red blood cells. The onset of the condition is triggered by temperatures 37ºC or higher. The severity of the disorder is variable.
Anemia, Neonatal: Insufficient red blood cells that can carry oxygen around the body. It is common in premature births or can occur as a result of blood loss before, during or just after the birth.
Anemia, Sideroblastic: A rare blood disorder where abnormal utilization of iron results in the production of defective red blood cells which have excessive deposits of iron in them.
Anemia, hypochromic microcytic: A blood disorder where red blood cells are too small and lack sufficient iron. It can be inherited or caused by insufficient iron in the diet or from a genetic disorder.
Anemias, Sideroblastic: Sideroblastic anemias are a group of rare blood disorders where the bone marrow is unable to produce normal red blood cells. The body has enough iron but the red blood cells are unable to utilize it in a normal manner and anemia results. The red blood cells become overloaded with iron and are unable to carry out their normal functions. Some forms of sideroblastic anemia are inherited but most tend to be acquired due to such things as exposure to toxins and certain drugs, leukemia, inflammatory conditions such as rheumatoid arthritis and nutritional deficiencies (e.g. copper and pyridoxine deficiency). Inherited forms usually appear in childhood whereas acquired forms usually occur in adulthood.
Anesthetic agent-induced liver damage: Damage or injury to the liver caused by exposure to anesthetic agents. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Anesthetic agent-induced liver damage -- Chloroform: Damage or injury to the liver caused by exposure to an anesthetic agent called chloroform. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Anesthetic agent-induced liver damage -- Cyclopropane: Damage or injury to the liver caused by exposure to an anesthetic agent called cyclopropane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Anesthetic agent-induced liver damage -- Ether: Damage or injury to the liver caused by exposure to an anesthetic agent called ether. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Anesthetic agent-induced liver damage -- Halothane: Damage or injury to the liver caused by exposure to an anesthetic agent called halothane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Anesthetic agent-induced liver damage -- Methoxyflurane: Damage or injury to the liver caused by exposure to an anesthetic agent called methoxyflurane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Anesthetic agent-induced liver damage -- Nitrous Oxide: Damage or injury to the liver caused by exposure to an anesthetic agent called nitrous oxide. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Angioimmunoblastic with dysproteinemia lymphadenopathy: A rare immune system disorder which is similar to lymphoma. The condition is progressive but the course varies with some patients surviving a long time without treatment and others surviving only a short period of time.
Angiosarcoma of the liver: A rare type of cancer that starts in the lining of blood vessels in the liver. It is generally an aggressive tumor which often metastasizes.
Antibiotics-induced liver damage: Damage or injury to the liver caused by taking certain antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antibiotics-induced liver damage -- Cephalosporin: Damage or injury to the liver caused by exposure to Cephalosporin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antibiotics-induced liver damage -- Chloramphenicol: Damage or injury to the liver caused by exposure to an antibiotic called Chloramphenicol. Chloramphenicol a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antibiotics-induced liver damage -- Clindamycin: Damage or injury to the liver caused by exposure to Clindamycin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antibiotics-induced liver damage -- Erythromycin Ethyl succinate: Damage or injury to the liver caused by exposure to Erythromycin Ethyl succinate antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antibiotics-induced liver damage -- Erythromycin estolate: Damage or injury to the liver caused by exposure Erythromycin estolate antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antibiotics-induced liver damage -- Nitrofuran: Damage or injury to the liver caused by exposure to Nitrofuran antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antibiotics-induced liver damage -- Novobiocin: Damage or injury to the liver caused by exposure to Novobiocin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antibiotics-induced liver damage -- Penicillin: Damage or injury to the liver caused by exposure to Penicillin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antibiotics-induced liver damage -- Quinolone: Damage or injury to the liver caused by exposure to Quinolone antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antibiotics-induced liver damage -- Rifampicin: Damage or injury to the liver caused by exposure to Rifampicin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antibiotics-induced liver damage -- Spectinomycin: Damage or injury to the liver caused by exposure to Spectinomycin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antibiotics-induced liver damage -- Sulfones: Damage or injury to the liver caused by exposure to sulfone antiboitics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antibiotics-induced liver damage -- Telithromycin: Damage or injury to the liver caused by exposure to Telithromycin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antibiotics-induced liver damage -- Tetracycline: Damage or injury to the liver caused by exposure to Tetracycline antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Anticonvulsive-induced liver damage: Damage or injury to the liver caused by exposure to anticonvulsives. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Anticonvulsive-induced liver damage -- Mephenytoin: Damage or injury to the liver caused by exposure to an anticonvulsive called mephenytoin. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Anticonvulsive-induced liver damage -- Phenobarbital: Damage or injury to the liver caused by exposure to an anticonvulsive called phenobarbital. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Anticonvulsive-induced liver damage -- Phenytoin: Damage or injury to the liver caused by exposure to an anticonvulsive called Phenytoin. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Anticonvulsive-induced liver damage -- Valproic Acid: Damage or injury to the liver caused by exposure to an anticonvulsive called valproic acid. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antifungal agent-induced liver damage: Damage or injury to the liver caused by exposure to antifungal agents. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antifungal agent-induced liver damage -- 5-Fluorocytosine: Damage or injury to the liver caused by exposure to an antifungal agent called 5-Fluorocytosine. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antifungal agent-induced liver damage -- Amphotericin: Damage or injury to the liver caused by exposure to an antifungal agent called Amphotericin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antifungal agent-induced liver damage -- Griseofulvin: Damage or injury to the liver caused by exposure to an antifungal agent called Griseofulvin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antifungal agent-induced liver damage -- Ketoconazole: Damage or injury to the liver caused by exposure to an antifungal agent called Ketoconazole. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antifungal agent-induced liver damage -- Saramycetin: Damage or injury to the liver caused by exposure to an antifungal agent called Saramycetin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antimetazoal agent-induced liver damage: Damage or injury to the liver caused by exposure to Antimetazoal agents. Antimetazoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antimetazoal agent-induced liver damage -- Amodiaquine: Damage or injury to the liver caused by exposure to an antimetazoal agent called amodiaquine. Antimetazoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antimetazoal agent-induced liver damage -- Hycanthone: Damage or injury to the liver caused by exposure to an antiprotozoal agent called hycanthone. Antimetazoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antiprotozoal agent-induced liver damage: Damage or injury to the liver caused by exposure to antiprotozoal agents. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antiprotozoal agent-induced liver damage -- 8-Hydroxyquinolone: Damage or injury to the liver caused by exposure to an antiprotozoal agent called 8-Hydroxyquinolone. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antiprotozoal agent-induced liver damage -- Carbarsone: Damage or injury to the liver caused by exposure to an antiprotozoal agent called carbarsone. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antiprotozoal agent-induced liver damage -- Emetine: Damage or injury to the liver caused by exposure to an antiprotozoal agent called emetine. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antiprotozoal agent-induced liver damage -- Mepacrine: Damage or injury to the liver caused by exposure to an antiprotozoal agent called mepacrine. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antiprotozoal agent-induced liver damage -- Metronidazole: Damage or injury to the liver caused by exposure to an antiprotozoal agent called Metronidazole. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antiprotozoal agent-induced liver damage -- Thiabendazole: Damage or injury to the liver caused by exposure to an antiprotozoal agent called Thiabendazole. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antituberculous agent-induced liver damage: Damage or injury to the liver caused by exposure to antituberculous agents. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antituberculous agent-induced liver damage -- Cycloserine: Damage or injury to the liver caused by exposure to an antituberculous agent called cycloserine. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antituberculous agent-induced liver damage -- Ethionamide: Damage or injury to the liver caused by exposure to an antituberculous agent called ethionamide. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antituberculous agent-induced liver damage -- Isoniazid: Damage or injury to the liver caused by exposure to an antituberculous agent called isoniazid. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antituberculous agent-induced liver damage -- Rifampicin: Damage or injury to the liver caused by exposure to an antituberculous agent called rifampicin. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antituberculous agent-induced liver damage -- p-aminosalicylic acid: Damage or injury to the liver caused by exposure to an antituberculous agent called p-aminosalicylic acid. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antiviral agent-induced liver damage: Damage or injury to the liver caused by exposure to antiviral agents. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antiviral agent-induced liver damage -- Cytarabine: Damage or injury to the liver caused by exposure to an antiviral agent called cytarabine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antiviral agent-induced liver damage -- Vidarabine: Damage or injury to the liver caused by exposure to an antiviral agent called vidarabine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antiviral agent-induced liver damage -- idoxuridine: Damage or injury to the liver caused by exposure to an antiviral agent called idoxuridine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Antiviral agent-induced liver damage -- xenylamine: Damage or injury to the liver caused by exposure to an antiviral agent called xenylamine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Aplastic anemia: A blood disorder where the bone marrow produces insufficient new blood cells.
Apo A-I deficiency: Low plasma HDL cholesterol that tends to run in families.
Arakawa's syndrome 2: An inherited metabolic disorder where an enzyme deficiency (methionine synthase) causes mental and physical retardation, blood disorders, degeneration of brain tissue and various other symptoms.
Arctic bearded seal poisoning: The Arctic Bearded seal is often used as a food source by the arctic inhabitants. Eating the liver and kidneys of the arctic bearded seal can result in a Vitamin A overdose which can cause serious symptoms and even death in extreme cases. It is believed that eating more than 100-250 grams of the seal liver can result in human death.
Arginase deficiency: A very rare urea cycle disorder caused by a deficiency of the enzyme (arginase) needed to convert ammonia to the urea which can then be removed in the urine. The condition leads to excess build-up of ammonia in the body which is toxic to the nervous system.
Argininosuccinase lyase deficiency, neonatal: A rare inherited urea cycle disorder caused by lack of enzymes (argininosuccinase lyase) needed to turn ammonia into urea resulting in excess ammonia in the body. The neonatal form of the condition can result in death or severe complications if not treated early enough.
Argininosuccinic aciduria: A rare inherited disorder of the urea cycle characterized by the lack of an enzyme (argininosuccinate lyase) which is needed to remove nitrogen from the body so a lack of the enzyme leads to a build-up of ammonia in the blood.
Arthralgia -- purpura -- weakness syndrome: A rare autoimmune disorder characterized mainly by weakness, joint pain, reddish or purplish skin discolorations and a blood abnormality where cryoglobulins in the blood precipitate at low temperatures.
Aspartylglucosaminidase deficiency: A rare glycoprotein metabolism disorder caused by a deficiency of an enzyme called aspartylglucosaminidase. Patients tend to develop normally during the first few years of life and development continues slowly until adolescence when mental retardation becomes progressively worse.
Aspartylglucosaminuria: A rare glycoprotein metabolism disorder caused by a deficiency of an enzyme called aspartylglucosaminidase. Patients tend to develop normally during the first few years of life and development continues slowly until adolescence when mental retardation becomes progressively worse.
Aspartylglycosaminuria: A rare glycoprotein metabolism disorder caused by a deficiency of an enzyme called aspartylglucosaminidase. Patients tend to develop normally during the first few years of life and development continues slowly until adolescence when mental retardation becomes progressively worse.
Asrar-Facharzt-Haque syndrome: A form of sinus histiocytosis characterized by lymphadenopathy as well as involvement of other tissues such as the sinus cavity, skin, lungs, bone, eyes, kidneys, testes, head and neck area and the central nervous system. Symptoms vary according to the organ involved. The condition usually runs a prolonged course with spontaneous remission occurring in some cases. Rare cases may be fatal.
Athabaskan severe combined immunodeficiency: A severe immunodeficiency disorder found in Navajo and Apache populations.
Australian Sea Lion poisoning: The Australian Sea Lion is sometimes used as a food source and is found in the South-Southwest waters of Australia. Eating the liver of the Australian Sea Lion can result in a Vitamin A overdose which can cause serious symptoms and even death in extreme cases.
Autoimmune Hemolytic Anemia: Autoimmune hemolytic anemia is characterized by an abnormal immune system response which leads to the destruction of red blood cells and hence anemia. The severity of the condition varies depending on the underlying cause e.g. cytomegalovirus, hepatitis, HIV and lupus. The condition may develop gradually or occur suddenly and cause serious symptoms.
Autoimmune Hepatitis: Liver inflammation caused due to autoimmune processes where the body's immune system attacks the liver.
Autoimmune Lymphoproliferative Syndrome: An inherited autoimmune condition characterized by proliferation of lymphocytes and autoimmunity against the body's own blood cells resulting in premature death of certain blood cells.
Autoimmune Thrombocytopenia: Autoimmune disorder causing a lack of blood platelets.
Autosomal Recessive Polycystic Kidney Disease: Severe form of PKD, a genetic kidney disease.
B-cell chronic lymphocytic leukemia: A slow progressing disease involving cancerous B-cell lymphocytes which take over the healthy cells in the lymph nodes. B-cells help the body to fight infections so when the disease becomes more advanced, the body is less able to fight infection as there are fewer healthy, functioning B-cells.
B-cell prolymphocytic leukaemia: A rare type of leukemia involving a proliferation of immature white blood cells (prolymphocytes - B-cells).
Baker-Winegrad disease: A very rare syndrome caused by a deficiency of the enzyme fructose-1-6-diphosphatase which impairs the body's ability to break down fructose that is consumed in the diet.
Ballard syndrome: A rare condition characterized by enlarged liver and spleen and fractures that occur in areas of weakened bone.
Banti Syndrome: A rare conditions where chronic congestive spleen enlargement causes it to destroy red blood cells too early. The spleen becomes enlarged due to an obstruction of blood flow in the organ and the resulting increase in blood pressure.
Banti's syndrome: A chronic, progressive condition marked by enlargement of the spleen which is associated with anemia, splenomegaly, ascites, jaundice, leucopenia, thrombocytopenia and gastrointestinal bleeding.
Bantu siderosis: An iron overload disorder initially observed in South African people. The disorder involves abnormal iron deposits in the liver. It is believed that some African people are predisposed to an increased ability to absorb iron.
Bartonella: A class of bacteria that can infect humans at a range of different sites. The most well known is Cat Scratch Disease, caused by B.henselae.
Bartonella infections: Infection with bacteria from the Bartonella genus of bacteria. Specific bacteria from within this group are Bartonella bacilliforms (Oroya fever), Bartonella Heneslae (Cat-scratch disease). Other conditions caused by this bacteria are endocarditis, bacteremia and angiomatosis. Symptoms vary depending on the type of bacteria involved and the severity of the infection - immunocompromised patients face greater risk of severe infection.
Bartonellosis: An infection by Bartonella bacilliformis which is transmitted through sandfly bites. Symptoms include fever, severe anemia, bone pain and skin lesions. Also called Carrion's disease, Oroya fever or verruga peruana.
Bartonellosis due to Bartonella quintana infection: A disease caused by infection with Bartonella quintana which are transmitted by the body louse. It causes trench fever but may also result in septicemia and endocarditis in patients with a weakened immune system.
Bearn-Kunkel syndrome: A type of autoimmune liver disease characterized by liver damage, very high blood gammaglobulin levels and increased plasma cells.
Benign intrahepatic cholestasis type 1: A rare liver disorder characterized by intermittent episodes of cholestasis (impaired bile flow within the liver) that doesn't involve obstruction of bile ducts outside the liver. The jaundice tends to disappear in weeks or months without treatment and the condition does not lead to liver failure. Type 1 is caused by a defect on the ATP8B1 gene on chromosome 18q21.
Benign intrahepatic cholestasis type 2: A rare liver disorder characterized by intermittent episodes of cholestasis (impaired bile flow within the liver) that doesn't involve obstruction of bile ducts outside the liver. The jaundice tends to disappear in weeks or months without treatment and the condition does not lead to liver failure. Type 2 is caused by a defect on the ABCB11 gene on chromosome 2q24.
Berardinelli-Seip congenital lipodystrophy: A rare genetic disorder characterized by diabetes mellitus, loss of body fat, hepatomegaly, enlarged genitals, increased skeletal growth and other abnormalities.
Berardinelli-Seip congenital lipodystrophy, type 1: A rare genetic disorder characterized by early-onset diabetes mellitus, loss of body fat, serious insulin resistance, high blood triglycerides and fatty liver. Type 1 is distinguished from type 2 by the origin of the genetic defect. Type 1 is caused by a defect on the AGPAT2 gene on chromosome 9q34.3. Type 1 seems to be less severe with some cases of type 2 resulting in premature death which can occur as early as the first year of life. Type 2 also involves mental retardation which is not seen in type 1.
Berardinelli-Seip congenital lipodystrophy, type 2: A rare genetic disorder characterized by early-onset diabetes mellitus, loss of body fat, serious insulin resistance, high blood triglycerides and fatty liver. Type 2 is distinguished from type 2 by the origin of the genetic defect. Type 2 is caused by a defect on the BSCL2 gene on chromosome 11q13. Type 2 seems to be more severe with some cases resulting in premature death which can occur as early as the first year of life. Type 2 also involves mental retardation which is not seen in type 1.
Bernheim's syndrome: Reduced size of right heart ventricle due to enlargement of the left ventricle which encroaches on the space in the right ventricle. Blood flow from the right atrium to the right ventricle is obstructed.
Besnier-Boeck-Schaumann disease: A rare disease where inflammatory granular nodules form in various organs.
Beta Thalassemia intermedia: Thalassemia is an inherited blood disorder characterized by abnormal synthesis of hemoglobin. There are two subtypes of the disorder (alpha and beta) depending on what portion of the hemoglobin is abnormally synthesized. Beta Thalassemia intermedia involves defects in both of the two genes required to make each ? protein chain. The condition causes varying degrees of moderate anemia.
Beta thalassemia: Thalassemia is an inherited blood disorder characterized by abnormal synthesis of hemoglobin. Hemoglobin consists of two main protein chains called alpha and beta. Beta thalassemia involves defects in one or more of the two genes required to make each ? protein chain. The main symptom is anemia, the severity of which can vary amongst patients depending on how many defective genes are involved.
Bile Duct Cancer: A malignancy arising in the bile ducts of the liver
Bile Duct Conditions: An inflammatory bacterial infection that affects the meninges
Bile acid synthesis defect, congenital, 2: A defect which prevents the body from making bile acid which results in progressive liver disease. The defect is a deficiency of a particular enzyme (cholestasis with delta(4)-3-oxosteroid 5-beta-reductase) needed to make bile acid.
Bile acid synthesis defect, congenital, 4: A defect which prevents the body from making bile acid which results in progressive liver disease.
Bile acid synthesis defects: A defect which prevents the body from making bile acid which results in progressive liver disease.
Bile acid synthesis defects, congenital, 1: A defect which prevents the body from making bile acid which results in progressive liver disease. The defect is a deficiency of a particular enzyme (3-beta-hydroxy-delta-5-C27-steroid oxidoreductase) needed to make bile acid.
Bile acid synthesis defects, congenital, 2: A defect which prevents the body from making bile acid which results in progressive liver disease. The defect is a deficiency of a particular enzyme (cholestasis with delta(4)-3-oxosteroid 5-beta-reductase) needed to make bile acid.
Bile acid synthesis defects, congenital, 3: A defect which prevents the body from making bile acid which results in progressive liver disease. The defect involved a deficiency of 7-alpha-hydroxylase which is an enzyme needed to prevent the accumulation of 27-hydroxycholesterol which is toxic to the liver.
Bile acid synthesis defects, congenital, 4: A defect which prevents the body from making bile acid which results in progressive liver disease.
Bile plug syndrome: A rare condition where thick bile in infants causes blockage of the bile ducts which can result in jaundice, enlarged liver and anemia.
Biliary Atresia: A rare condition that can affect newborns where the bile duct that carries the bile from the liver to the small intestine is blocked or missing.
Biliary atresia, intrahepatic, non syndromic form: Congenital obstruction of the passages in the liver that carry bile. The nonsyndromic form is not associated with any other abnormalities.
Biliary atresia, intrahepatic, syndromic form: Congenital obstruction of the passages in the liver that carry bile. The syndromic form is associated with other congenital abnormalities such as heart and visceral defects.
Biliary disorder: Any condition affecting the bile ducts
Biliary hypoplasia: An underdeveloped biliary duct system which is involved in transporting bile. The bile ductules may be completely absent or be fewer in number than normal.
Biotinidase deficiency, late onset: A metabolic disorder where the body lacks the enzyme biotinidase needed to process the vitamin called biotin (vitamin H) into carboxylase enzymes. The severity of symptoms may vary depending on the degree of deficiency. Severe cases can result in metabolic acidosis which can lead to death if treatment isn't given.
Birth symptoms: Symptoms related to childbirth.
Blastoma: A type of tumor that originates from precursor cells or blasts (immature or embryonic tissue). The symptoms can vary greatly and are determined by the part of the body that is affected. Blastomas can occur in parts of the body such as the brain, liver, kidneys, nervous system, bones and the retina.
Blastomycosis: A fungal infection caused by Blastomyces dermatitidis and resulting in lung, skin, bone and genitourinary involvement.
Boutonneuse fever: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
Breynia officinalis poisoning: Ingestion of the Breynia officinalis plant can cause irritation to mucosal linings and liver problems. The plant is often used as a herbal drug (Chi R Yun) to treat such things as poor growth, heart failure and venereal disease.
Brucellosis: An infectious disease caused by the Brucella genus which is transmitted from animals to humans.
Bruch's disease: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
Brunzell syndrome: A rare recessively inherited syndrome characterized by a lack of skin fat (Seip syndrome), cystic angiomatosis of the long bones which can lead to fractures.
Budd chiari syndrome: A disorder where the main vein leaving the liver becomes blocked leading to symptoms such as liver enlargement and fluid buildup in the abdomen. Also called Chiari's syndrome or Rokitansky's disease.
Burkholderia pseudomallei: Gram negative, aerobic, motile rod shaped bacterium.
Byler Disease: A rare inherited conditions where bile is unable to drain from the liver where it builds up and causes progressive liver damage. The conditions has an early onset and usually leads to end-stage liver disease by the end of the second decade.
CCF: When the heart is no longer able to pump enough blood to meet the needs of the body.
CDG syndrome (generic term): Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. The main symptom in all the disorders is psychomotor retardation but other variable symptoms also occur depending on the subtype of the disorder.
CDG syndrome type 1A: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1A involves a phosphomannomutase enzyme defect and affects most body systems especially the nervous system and liver function.
CDG syndrome type 1B: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1B has a phosphomannose isomerase enzyme defect.
CDG syndrome type 3: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 3 has variable symptoms.
CDG syndrome type I: A rare genetic disorder where the body is unable to synthesize glycoproteins which results in multisystem problems.
CML-Like Syndrome, Familial: A very rare condition characterized by symptom similar to myelocytic leukemia that develop during infancy. The condition can result in death during the first years of life.
CMV antenatal infection: A rare condition where a fetus becomes infected with the cytomegalovirus through the mother.
Camurati-Engelmann Disease: A very rare genetic disease thickening of areas of bone causing pain, weakness and wasting. Usually affects the legs during childhood. Also called diaphyseal dysplasia.
Carbamoyl-phosphate synthase 1 deficiency: A very rare inherited urea cycle disorder where the lack of the enzyme carbamoyl phosphate synthetase prevents ammonia from being turned into urea and being excreted in the urine. Excess ammonia builds up in the body which can cause serious complications or even death if left untreated.
Carcinoid syndrome: Carcinoid heart disease is a rare, metastatic disease that occurs predominantly in the right heart. The tricuspid and pulmonic valves are affected, leading to right heart failure, which results in increased morbidity and mortality.
Cardiomyopathy -- hypogonadism -- metabolic anomalies: A rare syndrome characterized mainly by heart muscle disease, hypogonadism, blindness, deafness and metabolic anomalies. Puberty was normal despite the hypogonadism.
Cardiomyopathy -- spherocytosis: A rare disorder characterized by the association of spherocytosis with heart muscle disease. Spherocytosis is a red blood cell disorder where the red blood cells have abnormal membranes which gives them a spherical shape and makes them weak resulting in their premature death.
Cardiomyopathy due to anthracyclines: Damage to the heart muscle caused by anthracycline drugs which are used in chemotherapy. The damage occurs more frequently with higher cumulative doses. Often the patients have no symptoms of the heart damage for many year.
Carnitine Palmitoyl Transferase I Deficiency: A very rare inherited deficiency of a particular enzyme (Carnitine palmitoyl transferase 1) prevents fatty acids being transported to the part of the cell that converts it to energy.
Carnitine Palmitoyl Transferase II Deficiency: A very rare inherited deficiency of a particular enzyme (Carnitine palmitoyl transferase 1) prevents fatty acids being transported to the part of the cell that converts it to energy. There are two main subtypes of the disorder with each involving a slightly different form of the enzyme. Type I can be readily managed through diet. Type II has three subtypes: the myopathic form affects mainly the muscles; the hepatocardiomuscular form affects the liver and heart muscle; and the lethal neonatal form affects muscles and organs and usually results in death during the first year of life.
Carnitine palmitoyl transferase 1 deficiency: A very rare inherited deficiency of a particular enzyme (Carnitine palmitoyl transferase I) prevents fatty acids being transported to the part of the cell that converts it to energy.
Carnitine palmitoyl transferase 2 deficiency: A very rare inherited deficiency of a particular enzyme (Carnitine palmitoyl transferase) which prevents fatty acids being transported to the part of the cell that converts it to energy. There are two main subtypes of the disorder with each involving a slightly different form of the enzyme. Type I can be readily managed through diet. Type II has three subtypes: the myopathic form affects mainly the muscles; the hepatocardiomuscular form affects the liver and heart muscle; and the lethal neonatal form affects muscles and organs and usually results in death during the first year of life.
Carnitine palmitoyl transferase II deficiency, infantile hepatocardiomuscular type: A very rare metabolic disorder where deficiency of a particular enzyme (CPT II) prevents muscle fats being converted to energy. The infantile form of this disease affects the muscles and the liver and heart.
Carnitine palmitoyl transferase deficiency: A very rare inherited deficiency of a particular enzyme (Carnitine palmitoyl transferase) which prevents fatty acids being transported to the part of the cell that converts it to energy. There are two main subtypes of the disorder with each involving a slightly different form of the enzyme. Type I can be readily managed through diet. Type II has three subtypes: the myopathic form affects mainly the muscles; the hepatocardiomuscular form affects the liver and heart muscle; and the lethal neonatal form affects muscles and organs and usually results in death during the first year of life.
Carnitine transporter deficiency: An inherited deficiency of carnitine caused by the impaired ability of the carnitine transporter protein to carry the carnitine to where it is needed. Instead the carnitine is excreted through the urine. Fasting or illness can trigger a severe attack.
Carnitine-acylcarnitine translocase deficiency: A very rare inherited metabolic disorder where long-chain fatty acids can't be metabolized properly because the compound needed to transport it is faulty. Ultimately this prevents certain fats (long-chain acylcarnitine) being converted to energy and results in a build up of the fat which is harmful to body organs and tissues.
Caroli Disease: A rare disorder where the bile ducts inside the liver become enlarged resulting in infection, irritation and gallstone formation.
Caspase-8 deficiency: A rare type of immunodeficiency disorder caused by a deficiency of caspase-8. Caspase-8 an important part of the immune system as it is involved in the activation of T lymphocytes, B lymphocytes and natural killer cells.
Castellani syndrome: A rare syndrome characterized by fever, arthritis and enlarged kidney and liver. The fever may fluctuate or come and go. It is most often seen in middle aged males.
Cephalosporin-induced Immune Hemolytic Anemia: Cephalosporin-induced immune hemolytic anemia is a condition where a use of a medication called Cephalosporin triggers the body's immune system to destroy it's own red blood cells which results in anemia.
Cerebrorenodigital syndrome: A rare group of syndromes characterized mainly by brain, kidney, finger and toe abnormalities.
Chagas disease: A parasitic infection caused by the protozoa Trypanosoma cruzi and transmitted by insect bites or blood transfusions. The disease primarily involves the heart and gastrointestinal system.
Chediak-Higashi Syndrome: An inherited immune system disorder resulting in frequent infections, lack of skin and eye pigmentation, neurological diseases and early death.
Chediak-Higashi like syndrome: A rare genetic disorder characterized mainly by albinism (lack of pigmentation). There are three different subtypes of the disorder (I, II and III) each with varying additional features such as immunodeficiency and neurological symptoms. Type 1 involves partial albinism and neurological symptoms, type II involves partial albinism, immunodeficiency and sometimes neurological symptoms and type III involves albinism only.
Chemical poisoning -- 1,2-Dibromoethane: 1,2-Dibromoethane is a chemical used in gasoline, soil fumigants, fire extinguishers, flue gases and mechanical gauge fluid. Excessive exposure to this chemical can cause serious symptoms. Some people can suffer an adverse reaction to the chemical. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
Chemical poisoning -- Chlorate salts: Chlorate salt is a chemical used mainly in herbicides and in the manufacture of matches and explosives. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
Chemical poisoning -- Chloroform: Chloroform is a chemical used mainly as a refrigerant but also as a solvent in various processing and industrial applications. It's use as an anesthetic is relatively uncommon these days. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
Chemical poisoning -- Dimethylnitrosamine: Dimethylnitrosamine is a chemical used mainly as a solving in the manufacture of plastics, rubbers, lubricants and rocket fuel. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
Chemical poisoning -- Methylene Dianiline: Methylene Dianiline is a chemical used mainly in corrosive inhibitors, epoxy resins and polyurethane. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
Chemical poisoning -- Phosphine: Phosphine is a chemical used mainly in pesticides and rodenticides. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
Chemical poisoning -- Tetrachloroethylene: Tetrachloroethylene is a chemical used mainly as a fabric dry cleaner, degreaser, worming treatment for animals and in the manufacture of freons. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
Chemical poisoning -- Vinyl Choride: Vinyl Choride is an intermediate chemical used in the manufacturing process of a variety of products: polyvinyl chloride, pipes, wire coverings, vehicle plastics, rubber, paper, furniture and glass. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
Chloroma: A rare type of tumor that develops from myeloid cells (type of immature white blood cell) and tends to usually have a greenish color. The tumor is essentially a localized solid collection of leukemic cells that occurs outside the bone marrow. It can occur in many parts of the body such as the chest, vertebrae, pelvis, skin, lymph nodes and parts of the skull. On rare occasions, it can occur in various organs such as the heart and brain. The tumor can occur as a primary condition or may be associated with cancerous conditions such as acute leukemia and acute promyelocytic leukemia. Symptoms will vary depending on the location of the tumor.
Chloromyeloma: A rare type of tumor that develops from myeloid cells (type of immature white blood cell) and tends to usually have a greenish color. The tumor is essentially a localized solid collection of leukemic cells that occurs outside the bone marrow. It can occur in many parts of the body such as the chest, vertebrae, pelvis, skin, lymph nodes and parts of the skull. On rare occasions, it can occur in various organs such as the heart and brain. The tumor can occur as a primary condition or may be associated with cancerous conditions such as acute leukemia and acute promyelocytic leukemia. Symptoms will vary depending on the location of the tumor.
Cholestasis -- lymphoedema, syndrome: A rare inherited birth abnormality involving underdeveloped lymph vessels which results in swollen legs and liver problems.
Cholestasis, progressive familial intrahepatic 1: A rare inherited condition where bile is unable to drain from the liver where it builds up and causes progressive liver damage.
Cholestasis, progressive familial intrahepatic 2: A rare inherited condition where bile is unable to drain from the liver where it builds up and causes progressive liver damage. The condition has an early onset and usually leads to end-stage liver disease by the end of the second decade. The various types of this condition differ in the origin of the genetic defect (liver-specific ATP-binding cassette transporter on chromosome 2q24). Type 2 is also associated with an increased risk of liver cancer in the first few years of life.
Cholestasis, progressive familial intrahepatic 3: A rare inherited condition where bile is unable to drain from the liver where it builds up and causes progressive liver damage. The condition has an early onset and usually leads to end-stage liver disease by the end of the second decade. The various types of this condition differ in the origin of the genetic defect (liver-specific ATP-binding cassette transporter on chromosome 2q24). Type 2 is also associated with an increased risk of liver cancer in the first few years of life.
Choriocarcinoma, infantile: A rare malignant cancer that originates in the placenta. It is an aggressive cancer which generally results in infant death.
Chromosome 11, deletion 11p: A syndrome that is caused by the deletion of short arm (p) of chromosome 11. The type and severity of symptoms varies depending on the amount and exact location of the genetic material that is deleted.
Chronic Granulomatous Disease: A very rare inherited blood disorder where certain cells involved with immunity (phagocytes) are unable to destroy bacteria and hence the patient suffers repeated bacterial infections.
Chronic Idiopathic Myelofibrosis: The bone marrow is consists of tissues which make blood cells and fibrous tissue supports these tissues that make the blood cells. In chronic idiopathic myelofibrosis, abnormal cells and fibres build up inside the bone marrow resulting in the production of fewer normal blood cells.
Chronic Lymphocytic Leukemia, Susceptibility to: A cancer of the blood and bone marrow where too many abnormal lymphocytes (white blood cells) are produced which eventually crowds out healthy blood cells in the body. Researchers have discovered a link between a number of genetic defects and increased susceptibility to chronic lymphocytic leukemia.
Chronic Lymphocytic Leukemia, Susceptibility to, 1: A cancer of the blood and bone marrow where too many abnormal lymphocytes (white blood cells) are produced which eventually crowds out healthy blood cells in the body. Researchers have discovered a link between a number of genetic defects and increased susceptibility to chronic lymphocytic leukemia. Type 1 is linked to a defect on chromosome 11q13.3
Chronic Lymphocytic Leukemia, Susceptibility to, 2: A cancer of the blood and bone marrow where too many abnormal lymphocytes (white blood cells) are produced which eventually crowds out healthy blood cells in the body. Researchers have discovered a link between a number of genetic defects and increased susceptibility to chronic lymphocytic leukemia. Type 2 is linked to a defect on chromosome 13q14.
Chronic Lymphocytic Leukemia, Susceptibility to, 3: A cancer of the blood and bone marrow where too many abnormal lymphocytes (white blood cells) are produced which eventually crowds out healthy blood cells in the body. Researchers have discovered a link between a number of genetic defects and increased susceptibility to chronic lymphocytic leukemia. Type 3 is linked to a defect on chromosome 9q34.1.
Chronic Lymphocytic Leukemia, Susceptibility to, 4: A cancer of the blood and bone marrow where too many abnormal lymphocytes (white blood cells) are produced which eventually crowds out healthy blood cells in the body. Researchers have discovered a link between a number of genetic defects and increased susceptibility to chronic lymphocytic leukemia. Type 4 is linked to a defect on chromosome 6p25.
Chronic Lymphocytic Leukemia, Susceptibility to, 5: A cancer of the blood and bone marrow where too many abnormal lymphocytes (white blood cells) are produced which eventually crowds out healthy blood cells in the body. Researchers have discovered a link between a number of genetic defects and increased susceptibility to chronic lymphocytic leukemia. Type 5 is linked to a defect on chromosome 11q24.1.
Chronic Myeloproliferative Disease, Unclassified: A form of blood disorder characterized by the abnormal proliferation of myeloid precursors in the bone marrow. This category refers to cases of myeloproliferative disease which don't fit into any of the other specific type of myelproliferative diseases.
Chronic Myeloproliferative Disorders: A group of blood cancers where excessive numbers of blood cells are made by overactive or cancerous bone marrow. The number of excess blood cells tends to grow slowly. Examples of such disorders includes chronic myelogenous leukemia, polycythemia vera and essential thrombocythemia. The symptoms are determined by which particular blood cancer is involved.
Chronic Neutrophilic Leukemia: A rare form of leukemia characterized by excessive levels of mature neutrophils.
Chronic Pesticide poisoning -- xylene: Xylene is an ingredient used in certain insecticides. Exposure to the chemical can cause a range of symptoms depending on the level and route of exposure. Exposure can occur through inhalation, ingestion, the skin or eyes. Acute exposure involves a exposure over a short period of time whereas chronic exposure occurs over a longer period of time.
Chronic adult T-Cell leukemia: A form of blood cancer affecting the T-cells which make up the body's immune system. The disease is caused by the HTLV-1 virus (human T-cell leukemia virus) which causes the proliferation of abnormal T-cells. The virus can be transmitted sexually and may lay dormant for decades. There are four subtypes: acute, chronic, lymphoma and smoldering. The acute and lymphoma subtypes have the poorest prognosis. The acute form tends to progress relatively slowly and generally responds better to treatment than the other subtypes.
Chronic fatigue syndrome: A persistent debilitating fatigue of recent onset
Chronic leukemia: Leukemia in which the cell line is well differentiated, usually B lymphocytes.
Chronic lymphocytic leukemia: A cancer of the blood and bone marrow where too many abnormal lymphocytes (white blood cells) are produced which eventually crowds out healthy blood cells in the body.
Chronic myelogenous leukemia: A slow-growing cancer of the white blood cells where the bone marrow makes too many white blood cells which eventually invade various parts of the body.
Chronic myeloid leukemia: Type of leukemia mostly in adults; rarely in children.
Chronic myelomonocytic leukemia: A rare form of malignant bone marrow cancer involving the proliferation of immature precursors of certain blood cells - myelocytes and monocytes. The proliferation is slower than in acute forms of the disease.
Chronic vitamin A toxicity: Chronic excessive ingestion of vitamin A can cause symptoms.
Cirrhosis, familial: Liver cirrhosis that is inherited in a familial pattern. The liver scarring (cirrhosis) is not caused by any discernable disease process. The liver becomes progressively scarred and its function is impaired.
Classic galactosemia: Rare serious genetic defect in galactose metabolism.
Classical Hodgkin disease: Hodgkin's disease is a type of cancer characterized by the abnormal proliferation of a type of white blood cell called lymphocyte. Hodgkin's lymphoma is classified into classical types and nodular lymphocyte predominant type. The nodular form tends to be more localized than the classical form. Classical Hodgkin's lymphoma is further subdivided into four subgroups depending on the cell composition of the lymphoma: nodular sclerosing, mixed cellularity, lymphocyte rich and lymphocyte depleted.
Cold Autoimmune Hemolytic Anemia: Cold autoimmune hemolytic anemia is a condition where the body's immune system triggers the production of antibodies against the body's own red blood cells. The red cells are destroyed at an abnormally rapid rate which leads to anemia. Cold haemolytic anemia is characterized by the fact that the abnormal destruction of red blood cells is more active when the patient is exposed to cold temperatures. The severity of the condition varies depending on the underlying cause. The condition may develop gradually or occur suddenly and cause serious symptoms.
Cold antibody hemolytic anemia: A rare autoimmune condition where the body's defense system attacks and destroys red blood cells. The onset of the condition is triggered by temperatures 30°C or lower.
Common Variable Immunodeficiency: An immunodeficiency disorder involving low blood gamma globulin levels which results in an increased susceptibility to infections. The condition may be inherited or can be caused by certain drugs (levamisole, hydantoin and carbamazepine).
Complex 1 mitochondrial respiratory chain deficiency: A rare genetic defect where an enzyme deficiency (NADH CoQ) disrupts cellular processes and causes various organic acid disorders. Any of a variety of the components of the enzyme may be missing or defective and hence the clinical presentation and severity may vary. Presentation may range from infantile death to various disorders such as Leigh's disease, Parkinson's disease and cardiomyopathy.
Complex 5 mitochondrial respiratory chain deficiency: A rare genetic defect where an enzyme deficiency (ATP synthetase) disrupts cellular processes. Any of a variety of the components of the enzyme may be missing or defective and hence the clinical presentation and severity may vary. The deficiency may result in a variety of symptoms and conditions of variable severity such as Leber's myopathy, Leigh syndrome, cardiomyopathy and NARP (neuropathy, ataxia, retinitis pigmentosa).
Congenital Disorder of Glycosylation, Type 1n: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1N has a defect in the RFT1 gene which results in decreased activity of an enzyme called dolichol-phosphage-mannose (Dol-P-M).
Congenital Disorders of Glycosylation Type Ia: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1A involves a phosphomannomutase enzyme defect and affects most body systems especially the nervous system and liver function.
Congenital Toxoplasmosis: Fetal infection with toxoplasmosis.
Congenital cytomegalovirus: Fetal infection with cytomegalovirus.
Congenital disorder of glycosylation type 1/IIX: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type I/IIX refers to cases where the specific abnormality cannot be determined.
Congenital disorder of glycosylation type 1A: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1A involves a phosphomannomutase enzyme defect and affects most body systems especially the nervous system and liver function.
Congenital disorder of glycosylation type 1B: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1B has a phosphomannose isomerase enzyme defect.
Congenital disorder of glycosylation type 1H: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type Ih is caused by a defect on chromosome 11pter-p15.5 and involves the gene for a particular enzyme (dolichyl-P-glucose:Glc-1-Man-9-GlcNAc-2-PP-dolichyl-alpha-3-glucosyltransferase).
Congenital disorder of glycosylation type 1I: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type Ii is caused by a defect on chromosome 9q22 and involves a defect on the ALG2 gene.
Congenital disorder of glycosylation type 1K: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type Ik is caused by a defect on chromosome 16p13.3 and involves a defect in the gene for beta-1,4-mannosyltransferase. The disorder is generally fatal within a year or two of birth.
Congenital disorder of glycosylation type 1L: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type Il is caused by a defect on chromosome 11q23 and involves a defect in the ALG9 gene.
Congenital disorder of glycosylation type 1X: Congenital disorder of glycosylation is a rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1X also involves thrombocytopenia with normal levels of phosphomannomutase and phosphomannose isomerase. This form of the condition is severe and results in death during infancy.
Congenital disorder of glycosylation type 2E: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 2e is caused by a defect on chromosome 16p and involves a defect in the gene for oligomeric complex-7.
Congenital disorder of glycosylation type 2G: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type IIg is caused by a defect on chromosome 17q25.1 and involves a defect on the COG1 gene.
Congenital disorder of glycosylation, type In: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1n involves a defect in the RFT1 gene and is characterized mainly by enlarged liver, seizures, developmental delay, reduced muscle tone and abnormal blood coagulation.
Congenital hepatic fibrosis: A rare inherited birth disorder characterized by fibrosis (scarring) of the liver which affects its ability to function. The severity of the condition is variable with some patients being symptomatic during infancy while others may be asymptomatic for most of their life.
Congenital hepatic porphyria: A rare congenital disorder where there is an excess of porphyrin (pigments) in the body. The liver is responsible for making porpyrins.
Congenital herpes simplex: An infant born with a herpes simplex infection transmitted through the mother. The infection may be localized or involve various internal organs and even the central nervous system in which case death can occur.
Congenital malaria: Fetal infection with malaria.
Congenital mumps: Fetal exposure to the mumps virus during pregnancy. From existing evidence, fetal exposure to mumps rarely proves to be a problem although there have been cases of spontaneous abortion and a heart condition called endocardial fibroelastosis. Infection late in the pregnancy can result in the infant contracting mumps.
Congenital spherocytic hemolytic anemia: A blood disorder present at birth where the membranes of red blood cells are defective which results in them being spherical rather than doughnut-shaped. These abnormally shaped red blood cells are broken down prematurely by the body which results in anemia. The condition may range from mild to severe.
Congenital tuberculosis: Fetal infection with tuberculosis
Conor's disease: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
Constrictive pericarditis: Constrictive pericarditis refers to an chronic inflammation of the sac that covers the heart (pericardium) that results in scarring of the pericardium.
Constrictive tuberculous pericarditis: Inflammation and swelling of the pericardium (fibrous sac surrounding the heart) that occurs as a complication of tuberculosis. The condition may be misdiagnosed as a heart attack and vice versa.
Cooley syndrome: An inherited blood disorder involving abnormal production of red blood cells which causes serious anemia. It is a severe form of thalassemia.
Cryoglobulinemia: These are immune cells that precipitate in the cold and redissolve on warming.
Cyclic neutropenia: A rare blood disorder characterized by recurrent periods of extremely low blood levels of neutrophils which results in frequent infections. The low level usually occurs for about 7 days every 21 days. Levels of other blood components may also be affected.
Cystic Fibrosis: Cystic fibrosis is a hereditary disease affecting the exocrine (mucus) glands of the lungs, liver, pancreas, and intestines, causing progressive disability due to multisystem failure.
Cytomegalic Inclusion Body Disease: An infection due to cytomegalovirus and marked by nuclear inclusion bodies in enlarged infected cells
Cytomegalovirus: A easily transmissible viral infection that is common but generally causes no symptoms except in infants and people with weakened immune systems.
DOC 12 (Neutral Lipid Storage Type): A rare inherited disorder involving the metabolism of fat which causes skin, muscle and blood abnormalities. The body is unable to metabolize (break down) triglycerides so they build up in various tissues throughout the body. The severity of the symptoms is variable as the body tissues and organs affected varies amongst patients.
Deafness -- lymphoedema -- leukemia: A rare syndrome characterized by deafness, early-onset leukemia and lymphoedema in the lower legs.
Deafness peripheral -- neuropathy -- arterial disease: A very rare syndrome characterized mainly by artery disease, deafness and peripheral neuropathy.
Debler anemia: A familial type of anemia that starts during infancy and involves the premature destruction of red blood cells.
Defect in synthesis of adenosylcobalamin: A rare genetic disorder characterized by the impaired ability to make a chemical called adenosylcobalamin. Adenosylcobalamin is a derivative of vitamin B12. The defect results a biochemical abnormality which affects the body's normal biochemical functioning.
Deletion 11p: A syndrome that is caused by the deletion of short arm (p) of chromosome 11. The type and severity of symptoms varies depending on the amount and exact location of the genetic material that is deleted.
Di Guglielmo I -- acute: A rare condition characterized by the presence of abnormal red blood cells in the bone marrow and blood. The condition is characterized by anemia and generally leads to the development of acute myelogenous leukemia. The acute form has more severe symptoms than the chronic form.
Di Guglielmo I -- chronic: A rare condition characterized by the presence of abnormal red blood cells in the bone marrow and blood. The condition is characterized by anemia and generally leads to the development of acute myelogenous leukemia. The chronic form has more severe symptoms than the acute form.
Dianzani autoimmune lymphoproliferative syndrome: A rare inherited disorder where the body's immune system attack itself, primarily blood components.
Dibasic aminoaciduria 2: A rare condition where protein intolerance occurs as a result of a defect in the transport of dibasic amino acids through the intestines and kidneys. The amino acids (component of protein) can't be broken down properly and used by the body so it builds up and causes damage.
Diffuse neonatal hemangiomatosis: A very rare syndrome characterized mainly by multiple capillary hemangiomas as well as other abnormalities.
Digestive symptoms: Any symptoms affecting the digestive tract.
Disorder of Cornification 12 (Neutral Lipid Storage Type): A rare inherited disorder involving the metabolism of fat which causes skin, muscle and blood abnormalities. The body is unable to metabolize (break down) triglycerides so they build up in various tissues throughout the body. The severity of the symptoms is variable as the body tissues and organs affected varies amongst patients.
Disorders of the spleen in young adults:
Distomatosis: Infection by parasitic flat worms. Infection can involve liver, lungs or intestines. Symptoms are determined by the location of the infection. Contamination usually occurs through ingesting contaminated food or water.
Doxepine-induced Immune Hemolytic Anemia: Doxepine -induced immune hemolytic anemia is a condition where use of a medication called Methyldopa triggers the body's immune system to destroy it's own red blood cells which results in anemia.
Dressler (D.)syndrome: A rare autoimmune blood disorder where erythrocytes are destroyed suddenly after exposure to cold (usually 15°C or lower).
Drug-induced Immune Hemolytic Anemia: Drug-induced immune hemolytic anemia is a condition where a medication triggers the body's immune system to destroy its own red blood cells which results in anemia. Certain drugs are more likely to trigger this abnormal immune response than others e.g. cephalosphorins and Quinidine.
Drug-induced liver damage -- 5-Fluorocytosine: Damage or injury to the liver caused by exposure to an antifungal agent called 5-Fluorocytosine. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Allopurinol: Damage or injury to the liver caused by exposure to allopurinol. Allopurinol is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Amphotericin: Damage or injury to the liver caused by exposure to an antifungal agent called Amphotericin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Anabolic C-17: Damage or injury to the liver caused by exposure to Anabolic C-17 which is an endocrine agent. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Anesthetic agent: Damage or injury to the liver caused by exposure to anesthetic agents. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Antianginal agents: Damage or injury to the liver caused by exposure to antianginal agents. Antianginal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Antiarrhythmics: Damage or injury to the liver caused by exposure to antiarrhythmics. Antiarrhythmics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Antibiotics: Damage or injury to the liver caused by exposure to certain antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Anticoagulants: Damage or injury to the liver caused by exposure to anticoagulants. Anticoagulants are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Antifungals: Damage or injury to the liver caused by exposure to antifungal agents. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Antihyperlipidemic agents: Damage or injury to the liver caused by exposure to antihyperlipidemic agents. Antihyperlipidemic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Antihypertensives: Damage or injury to the liver caused by exposure to antihypertensives. Antihypertensives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Antineoplastic agents: Damage or injury to the liver caused by exposure to antineoplastic agents. Antineoplastic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Antithyroid drugs: Damage or injury to the liver caused by exposure to endocrine agents called antithyroid drugs. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Benzodiazepine: Damage or injury to the liver caused by exposure to a psychotropic agent called benzodiazepine. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- British anti-Lewisite penicillamine: Damage or injury to the liver caused by exposure to British anti-Lewisite penicillamine. British anti-Lewisite penicillamine is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Butyrophenone: Damage or injury to the liver caused by exposure to a psychotropic agent called butyrophenone. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Cephalosporin: Damage or injury to the liver caused by exposure to Cephalosporin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Chloramphenicol: Damage or injury to the liver caused by exposure to Chloramphenicol. Chloramphenicol is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Chloroform: Damage or injury to the liver caused by exposure to an anesthetic agent called chloroform. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Cimetidine: Damage or injury to the liver caused by exposure to Cimetidine. Cimetidine is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Clindamycin: Damage or injury to the liver caused by exposure to Clindamycin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Colchicine: Damage or injury to the liver caused by exposure to colchicine. Colchicine is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Cyclopropane: Damage or injury to the liver caused by exposure to an anesthetic agent called cyclopropane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Cycloserine: Damage or injury to the liver caused by exposure to an antituberculous agent called cycloserine. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Cytarabine: Damage or injury to the liver caused by exposure to an antiviral agent called cytarabine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Dantrolene: Damage or injury to the liver caused by exposure to Dantrolene. Dantrolene is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Diflunisal: Damage or injury to the liver caused by exposure to diflunisal. Diflunisal is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Disulfiram: Damage or injury to the liver caused by exposure to Disulfiram. Disulfiram is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Diuretic Agents: Damage or injury to the liver caused by exposure to diuretic agents. Diuretic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Erythromycin estolate: Damage or injury to the liver caused by exposure to an antibiotic called erythromycin estolate. Erythromycin estolate is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Erythromycin ethyl succinate: Damage or injury to the liver caused by exposure to an antibiotic called Erythromycin ethyl succinate. Erythromycin ethyl succinate is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Ethionamide: Damage or injury to the liver caused by exposure to an antituberculous agent called ethionamide. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Fenoprofen: Damage or injury to the liver caused by exposure to fenoprofen. Fenoprofen is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Glucocorticoids: Damage or injury to the liver caused by exposure to endocrine agents called glucocorticoids. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Griseofulvin: Damage or injury to the liver caused by exposure to an antifungal agent called Griseofulvin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Halothane: Damage or injury to the liver caused by exposure to an anesthetic agent called halothane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Ibuprofen: Damage or injury to the liver caused by exposure to ibuprofen. Ibuprofen is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Indomethacin: Damage or injury to the liver caused by exposure to indomethacin. Indomethacin is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Iodide ion: Damage or injury to the liver caused by exposure to Iodide ion. Iodide ion is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Isoniazid: Damage or injury to the liver caused by exposure to an antituberculous agent called isoniazid. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Ketoconazole: Damage or injury to the liver caused by exposure to an antifungal agent called Ketoconazole. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Mephenytoin: Damage or injury to the liver caused by exposure to an anticonvulsive called mephenytoin. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Methoxyflurane: Damage or injury to the liver caused by exposure to an anesthetic agent called methoxyflurane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Naproxen: Damage or injury to the liver caused by exposure to naproxen. Naproxen is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Nitrofuran: Damage or injury to the liver caused by exposure to Nitrofuran antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Nitrous Oxide: Damage or injury to the liver caused by exposure to an anesthetic agent called nitrous oxide. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Novobiocin: Damage or injury to the liver caused by exposure to Novobiocin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Oral hypoglycemics: Damage or injury to the liver caused by exposure to oral hypoglycemics which are endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Penicillin: Damage or injury to the liver caused by exposure to an antibiotic called penicillin. Penicillin is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Phenobarbital: Damage or injury to the liver caused by exposure to an anticonvulsive called phenobarbital. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Phenothiazines: Damage or injury to the liver caused by exposure to a psychotropic agent called phenothiazine. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Phenylbutazone: Damage or injury to the liver caused by exposure to phenylbutazone. Phenylbutazone is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Phenytoin: Damage or injury to the liver caused by exposure to an anticonvulsive called Phenytoin. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Quinolone: Damage or injury to the liver caused by exposure to Quinolone antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Ranitidine: Damage or injury to the liver caused by exposure to Ranitidine. Ranitidine is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Rifampicin: Damage or injury to the liver caused by exposure to an antituberculous agent called rifampicin. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Salicylate: Damage or injury to the liver caused by exposure to salicylates. Salicylates are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Saramycetin: Damage or injury to the liver caused by exposure to an antifungal agent called Saramycetin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Spectinomycin: Damage or injury to the liver caused by exposure to Spectinomycin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Steroids: Damage or injury to the liver caused by exposure to steroids which are endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Sulfonamide: Damage or injury to the liver caused by exposure to Sulfonamide antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Sulfones: Damage or injury to the liver caused by exposure to sulfone antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Sulindac: Damage or injury to the liver caused by exposure to sulindac. Sulindac is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Tamoxifen: Damage or injury to the liver caused by exposure to Tamoxifen which is an endocrine agent. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Telithromycin: Damage or injury to the liver caused by exposure to an antibiotic called Telithromycin. Telithromycin is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Tetracycline: Damage or injury to the liver caused by exposure to Tetracycline antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Thioxanthene: Damage or injury to the liver caused by exposure to a psychotropic agent called Thioxanthene. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Thorotrast: Damage or injury to the liver caused by exposure to Thorotrast. Thorotrast is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Valproic Acid: Damage or injury to the liver caused by exposure to an anticonvulsive called valproic acid. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Vidarabine: Damage or injury to the liver caused by exposure to an antiviral agent called vidarabine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Vitamin A: Damage or injury to the liver caused by exposure to Vitamin A. Vitamin A is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- Zoxazolamine: Damage or injury to the liver caused by exposure to Zoxazolamine. Zoxazolamine is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- anticonvulsives: Damage or injury to the liver caused by exposure to certain anticonvulsives. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- antituberculous agents: Damage or injury to the liver caused by exposure to antituberculous agents. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- antiviral medication: Damage or injury to the liver caused by exposure to antiviral agents. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- endocrine agent: Damage or injury to the liver caused by exposure to endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- idoxuridine: Damage or injury to the liver caused by exposure to an antiviral agent called idoxuridine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- monoamine oxidase inhibitors: Damage or injury to the liver caused by exposure to psychotropic agents called monoamine oxidase inhibitors. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- p-aminosalicylic acid: Damage or injury to the liver caused by exposure to an antituberculous agent called p-aminosalicylic acid. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- psychotropic agents: Damage or injury to the liver caused by exposure to certain psychotropic agents. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- tricyclic antidepressant: Damage or injury to the liver caused by exposure to a psychotropic agent called tricyclic antidepressant. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drug-induced liver damage -- xenylamine: Damage or injury to the liver caused by exposure to an antiviral agent called xenylamine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Drugs-induced liver damage -- Ether: Damage or injury to the liver caused by exposure to an anesthetic agent called ether. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Dyck Syndrome: A very rare condition observe in two brothers. It involves vision, hearing, muscle, sensory and adrenal system problems and an enlarged liver and spleen.
Dykes-Markes-Harper syndrome: A very rare syndrome characterized mainly by dry, scaly skin, enlarged liver and spleen and a incoordination.
Dyserythropoietic anemia, congenital: A congenital blood condition where the production of red blood cells is defective. The abnormal red blood cells are often destroyed prematurely within the body which can result in anemia. There are three subtypes of the disorder, each with varying severity of symptoms.
Dyserythropoietic anemia, congenital type 1: A very rare genetic blood disorder where abnormal red blood cells are made resulting in anemia. Finger and toe abnormalities are also usually present.
Dyserythropoietic anemia, congenital type 2: A very rare genetic blood disorder where abnormal red blood cells are made resulting in anemia.
Dyserythropoietic anemia, congenital type 3: An extremely rare blood disorder where abnormal red blood cells are made resulting in anemia.
Dysplastic cortical hyperostosis: A very rare syndrome characterized mainly by abnormal bone and brain development.
Eccentrochondrodysplasia: A rare inherited biochemical disorder characterized by the accumulation of mucopolysaccharides (glycosaminoglycans) in various body tissues due to insufficient amounts of certain enzymes needed to break it down.
Ectodermal dysplasia, anhidrotic with T-cell immunodeficiency, autosomal dominant: An inherited disorder characterized by dry, rough skin, sparse scalp hair, cone-shaped teeth and an immune system disorder.
Elejalde syndrome: A rare congenital syndrome characterized excessive fetal growth, skull abnormalities, extra fingers or toes and other abnormalities.
Emanuel syndrome: A rare chromosomal disorder where a portion of chromosome 11 is translocated with a portion of chromosome 22. The disorder involves a wide range of abnormalities such as kidney, genital and heart abnormalities, mental retardation, small head and failure to thrive.
Encephalopathy due to GLUT1 deficiency: A rare inherited metabolic disorder where a genetic mutation results in the deficiency of an enzyme called glutaryl-CoA dehydrogenase which is required to metabolise certain amino acids (lysine, hydroxylysine and tryptophan). Problems occur when these metabolites build up in the body and cause neurological problems. Symptoms often develop following an acute infection or fasting. The severity of the condition is highly variable from development of neurological symptoms during infancy to asymptomatic adults. The degree of enzyme deficiency will usually determine the severity.
End Stage Liver Failure: Late stage of liver failure characterised by the onset of mental and neurological symptoms, due to build up of toxic metabolites.
Endocardial fibroelastosis: A rare heart disorder characterized by a thickening of the heart muscle lining causing heart enlargement and heart failure.
Endocrine agent-induced liver damage: Damage or injury to the liver caused by exposure to endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Endocrine agent-induced liver damage -- Anabolic C-17: Damage or injury to the liver caused by exposure to Anabolic C-17 which is an endocrine agent. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Endocrine agent-induced liver damage -- Antithyroid drugs: Damage or injury to the liver caused by exposure to endocrine agents called antithyroid drugs. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Endocrine agent-induced liver damage -- Glucocorticoids: Damage or injury to the liver caused by exposure to endocrine agents called glucocorticoids. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Endocrine agent-induced liver damage -- Oral contraceptives: Damage or injury to the liver caused by exposure to oral contraceptives which are endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Endocrine agent-induced liver damage -- Oral hypoglycemics: Damage or injury to the liver caused by exposure to oral hypoglycemics which are endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Endocrine agent-induced liver damage -- Steroids: Damage or injury to the liver caused by exposure to steroids which are endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Endocrine agent-induced liver damage -- Tamoxifen: Damage or injury to the liver caused by exposure to Tamoxifen which is an endocrine agent. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
Endomyocardial fibrosis: Heart disease characterized by progressive fibrosis of the lining of one or both lower heart cavities. This results in constriction of the heart cavity and may involve the heart valves and other structures.
Enlarged liver: Swelling of the liver.
Enlarged liver and spleen: Enlargement of both liver and spleen (hepatosplenomegaly)
Enlarged spleen in children: An enlarged spleen in children is a swelling or distention of the spleen in a child.
Enterovirus antenatal infection: Fetal infection with enterovirus. The condition is extremely rare but infection around the time of birth often results in death or paralysis in survivors. The type and severity of symptoms is determined by the exact type of virus involved and at what stage of development the infection occurs.
Epstein-Barr virus, chronic: A form of human herpes virus that produces persistent symptoms. Most people become infected with the virus at some stage in their life though they usually have few if any symptoms. However, some people develop severe symptoms as a result of an EBV infection.
Erdheim-Chester Disease: A condition which is defined as a non langerhans cell histiocytosis
Erdheim-Chester syndrome: A very rare lipid storage disorder involving lipid deposits in various organs and hardening of the ends of long bones which affects the growth of the bone. The severity of the condition is variable.
Escharonodulaire: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
Essential mixed cryoglobulinemia: A rare autoimmune disorder characterized by the presence of a mixture of various types of antibodies (crylglobulins) in the blood.
Essential thrombocytosis -- same as essential thrombocythemia: A rare blood disorder where the blood contains too many platelets due to excessive megakaryocytes (platelet-producing cells). Platelets are essential for blood clotting but in essential thrombocythemia excessive platelets can cause the blood to form abnormal clots. If the platelets are defective as well then bleeding problems can occur. The severity of the condition is variable.
Exocrine Pancreatic Insufficiency, Dyserythropoietic Anemia, And Calvarial Hyperostosis: A rare syndrome characterized mainly by anemia, pancreatic dysfunction and abnormal skull calcification.
Extramedullary Myeloid Tumor: A rare type of tumor that develops from myeloid cells (type of immature white blood cell) and tends to usually have a greenish color. The tumor is essentially a localized solid collection of leukemic cells that occurs outside the bone marrow. It can occur in many parts of the body such as the chest, vertebrae, pelvis, skin, lymph nodes and parts of the skull. On rare occasions, it can occur in various organs such as the heart and brain. The tumor can occur as a primary condition or may be associated with cancerous conditions such as acute leukemia and acute promyelocytic leukemia. Symptoms will vary depending on the location of the tumor.
Familial Lipoprotein Lipase Deficiency: An hereditary condition characterized by a deficiency of lipoprotein lipase
Familial Mediterranean fever: A rare inherited condition characterized by recurrent fever and inflammation. The inflammation usually involves the stomach, lungs or joints.
Familial hyperchylomicronemia: A rare inherited inborn error of metabolism involving the absence of the enzyme called lipoprotein lipase which results in increased blood triglyeride and chylomicron levels.
Familial hyperlipoproteinemia: A group of genetic disorder characterized by abnormal breakdown of lipoproteins which causes abnormal lipoprotein and lipid levels in the blood. There are various types of this condition: hyperlipoproteinemia type I, II, III, IV and V. The type and severity of symptoms vary between types. The disorder tends to run in families (familial).
Familial hyperlipoproteinemia type 1: A genetic disorder characterized by abnormal lipid (chylomicrons and high triglyceride lipids) breakdown which results in its accumulation in the blood. The disorder is caused by the reduced or absent activity of the enzyme lipoprotein lipase. The severity of the condition is determined by the degree of the deficiency and treatment. The disorder tends to run in families (familial).
Familial myelofibrosis: A rare condition where progressive scarring or fibrosis of the bone marrow impairs it's ability to make blood cells causing symptoms such as anemia and liver and spleen enlargement.
Fanconi-Bickel syndrome: A rare inherited disorder where the impaired metabolism of carbohydrates results in a build-up of glycogen in the liver.
Farber Disease: A lysosomal storage disease due to defective ceramidase and characterized by hoarsness, aphonia , dermatitis and psychomotor retardation.
Farber's disease: A rare inherited biochemical disorder involving the deficiency of an enzyme called ceramidase resulting in the harmful accumulation of certain chemicals in the body which causes damage and inflammation.
Fascioliasis: A rare parastitic infectious disease caused by liver fluke Fasciola hepatica which can cause blockage of the bile ducts in the liver.
Felty syndrome: A rare complication of long-term rheumatoid arthritis which involves an enlarged spleen and blood abnormalities.
Fluke infections: An infection caused by flukes
Forbes disease: A rare inherited glycogen storage disease caused by a deficiency of the enzyme amylo-1,6-glucosidase resulting in a build up of glycogen in the liver and muscles.
Fowl Paralysis: A viral disease that affects poultry - it is very contagious and is a herpes virus. The virus tends to affect the nerves and cause tumors in internal organs. Poultry may be unable to lay eggs or may even die. Young birds are more susceptible than older birds with death most common between the ages of 8 and 20 weeks. The virus may attack the nervous system and result in paralysis or it may attack the visceral system and cause tumors in the internal organs. Some birds may die without any obvious symptoms.
Fowl paralyses: A viral disease that affects poultry - it is very contagious and is a herpes virus. The virus tends to affect the nerves and cause tumors in internal organs. Poultry may be unable to lay eggs or may even die. Young birds are more susceptible than older birds with death most common between the ages of 8 and 20 weeks. The virus may attack the nervous system and result in paralysis or it may attack the visceral system and cause tumors in the internal organs. Some birds may die without any obvious symptoms.
Free sialic Acid storage disease: A rare inherited biochemical disorder characterized by the accumulation of sialic acid in the tissues and excretion of sialic acid in the urine. There are mild and severe forms of the condition - the severe form result in death before birth or within a few years of birth.
Fructose intolerance: An enzyme deficiency that causes toxic symptoms when fructose containing food is ingested.
Fructose-1,6-bisphosphatase deficiency, hereditary: A rare inherited condition where an enzyme deficiency (fructose-1,6-bisphosphatase deficiency - FDPase) impairs the body's ability to metabolize fructose from the diet.
Fructose-1-phosphate aldolase deficiency, hereditary: An inherited metabolic disorder where deficiency of the enzyme fructose-1-phsophate aldolase prevents fructose being metabolized resulting in fructose intolerance.
Fucosidosis type 1: A rare biochemical disorder involving deficiency of an enzyme (alpha-fucosidase) which results in accumulation of certain chemicals (glycosphingolipids) in the central nervous system and other body tissues. It is an infantile form of fucosidosis which starts early and rapidly progresses to early death.
Fucosidosis type II: A form of the biochemical disorder called fucosidosis where an enzyme deficiency (alpha-fucosidase) results in the accumulation of certain chemicals (glycosphingolipids) in the central nervous system and other body tissues. Symptoms start later and progress slower than in type I and is distinguished by warty skin growths.
GM1 gangliosidosis: A rare biochemical disorder involving a deficiency of an enzyme (beta-galactosidase A) which results in the accumulation of harmful chemicals (GM1 gangliosides) in the central nervous system and other body tissues. Type 1 is a severe infantile form of the disorder and involves a greater degree of accumulation than type II or III.
Galactokinase deficiency: A rare condition where an enzyme deficiency (galactokinase) impaires the body's ability to break down galactose consumed in the diet.
Galactosemia I: A rare inherited disorder where deficiency of a particular enzyme (galactose-1-phosphate uridyl transferase) prevents the metabolism of galactose which is a sugar component of milk. Ranges from milk intolerance in mild cases to death in severe untreated cases.
Gall Bladder Cancer: Cancer of the gall bladder.
Gall bladder conditions: Any condition that affects the gallbladder
Game-Friedman-Paradice syndrome: A rare condition characterized by retarded growth, hydrocephaly, underdeveloped lungs and various other anomalies. The condition was observed in four offspring from one family.
Gangliosidosis generalized GM1, type 1: A rare biochemical disorder involving a deficiency of an enzyme (beta-galactosidase A) which results in the accumulation of harmful chemicals (GM1 gangliosides) in the central nervous system and other body tissues. Type 1 is a severe infantile form of the disorder and involves a greater degree of accumulation than type II or III.
Garret-Tripp syndrome: A rare syndrome characterized mainly by extra digits, a lack of hair and scaly skin on the scalp, face and neck.
Gaucher Disease: A rare inherited biochemical disorder characterized by the deficiency of the enzyme called glucocerebrosidase and accumulation of glycosylceramide (glucocerebroside). There are three forms of this disease: type 1, 2 and 3.
Gaucher disease -- perinatal lethal form: A rare syndrome characterized by the association of abnormally tight skin and Gaucher disease which is a lipid storage disease. This is the most severe form of Gaucher disease.
Gaucher disease type 1: A rare inherited biochemical disorder characterized by the deficiency of the enzyme called glucocerebrosidase and accumulation of glycosylceramide (glucocerebroside). There are three forms of this disease: type 1, 2 and 3. Type 1 is the visceral, chronic form which usually starts during adulthood.
Gaucher disease type 2: A rare inherited biochemical disorder characterized by the deficiency of the enzyme called glucocerebrosidase and accumulation of glycosylceramide (glucocerebroside). There are three forms of this disease: type 1, 2 and 3. Type 2 is acute neurological form apparent in infancy.
Gaucher disease type 3: A rare inherited biochemical disorder characterized by the deficiency of the enzyme called glucocerebrosidase and accumulation of glycosylceramide (glucocerebroside). There are three forms of this disease: type 1, 2 and 3. Type 3 is a subacute neurological form which often first appears in childhood.
Gaucher-like disease: A very rare inherited condition where the body's inability to bread down a certain fat (glucocerebroside) causes it to accumulate in body tissues and organs where it can cause damage. Gaucher-like disease is a sub-type of Gaucher disease which primarily affects the heart.
Geleophysic dwarfism: A rare genetic disorder characterized by a happy facial expression, short stature and limb abnormalities.
Ghosal syndrome: A very rare syndrome characterized mainly by difficult to treat anemia and skeletal abnormalities.
Glanders: An infectious disease caused by a bacterium (Burkholderia mallei). It is usually a disease that affects horses and mules but can also infect other animals and humans. Human infection usually occurs in laboratory settings or in those with prolonged contact with infected animals. Symptoms are determined by whether infection occurs through the skin or via the lungs or blood stream. Bloodstream infections are the most severe and usually result in death within weeks.
Glutaric Acidemia Type I: A condition which results in an inability to process the amino acids lysine, hydroxylysine and tryptophan
Glutaric Acidemia Type II: A condition which is characterized by an inability of the body to use fats and proteins of the body for energy
Glutaric Aciduria, neonatal form of type II A: A more serious neonatal form of glutaricaciduria where there is excessive blood and urine levels of glutaric acid and congenital anomalies may be present.
Glutaric aciduria 1: A rare inherited metabolic disorder where a genetic mutation results in the deficiency of an enzyme called glutaryl-CoA dehydrogenase which is required to metabolise certain amino acids (lysine, hydroxylysine and tryptophan). Problems occur when these metabolites build up in the body and cause neurological problems. Symptoms often develop following an acute infection or fasting. The severity of the condition is highly variable from development of neurological symptoms during infancy to asymptomatic adults. The degree of enzyme deficiency will usually determine the severity.
Glutaric aciduria 2: A metabolic disorder involving an enzyme deficiency - electron transfer flavoprotein ubiquinone oxydoreductase. The severity of symptoms depends on the level of deficiency. The infant onset form is the most severe and often results in death. Severe cases usually develop during childhood or infancy and usually involve metabolic acidosis and its associated symptoms. Milder cases may simply present with muscle weakness initially that develops in adulthood. Some cases may involve additional symptoms such as heart, liver and kidney problems, facial anomalies and genital abnormalities.
Glutaricaciduria 2B: A milder, later-onset form of glutaricaciduria where there is excessive blood and urine levels of glutaric acid due to the body's impaired ability to metabolize protein and fat into energy.
Glutaricaciduria I: A rare inherited enzyme deficiency disorder where deficiency of the glutaryl-CoA dehydrogenase enzyme results in dystonia, dyskinesia and sometimes mental retardation.
Glutaricaciduria type 1: A rare inherited metabolic disorder where a genetic mutation results in the deficiency of an enzyme called glutaryl-CoA dehydrogenase which is required to metabolise certain amino acids (lysine, hydroxylysine and tryptophan). Problems occur when these metabolites build up in the body and cause neurological problems. Symptoms often develop following an acute infection or fasting. The severity of the condition is highly variable from development of neurological symptoms during infancy to asymptomatic adults. The degree of enzyme deficiency will usually determine the severity.
Glutaryl-CoA dehydrogenase deficiency: A rare inherited metabolic disorder where a genetic mutation results in the deficiency of an enzyme called glutaryl-CoA dehydrogenase which is required to metabolise certain amino acids (lysine, hydroxylysine and tryptophan). Problems occur when these metabolites build up in the body and cause neurological problems. Symptoms often develop following an acute infection or fasting. The severity of the condition is highly variable from development of neurological symptoms during infancy to asymptomatic adults. The degree of enzyme deficiency will usually determine the severity.
Glycogen Storage Disease IXa1: Glycogen storage disease type IX is a relatively mild glycogen storage disease which involves a deficiency of the enzyme hepatic phosphorylase kinase. Thee are four subtypes of the condition, each caused by a different genetic defect which results in the enzyme deficiency. Type IXa is linked to a defect in the PHKA2 gene on chromosome Xp22.2-p22.1. It is inherited in a X-linked recessive manner which means that only males will exhibit symptoms though females may be carriers.
Glycogen Storage Disease IXb: Glycogen storage disease type IX is a relatively mild glycogen storage disease which involves a deficiency of the enzyme hepatic phosphorylase kinase. There are four subtypes of the condition, each caused by a different genetic defect which results in the enzyme deficiency. Type IXb is linked to a defect in the PHKG2 gene on chromosome 16q12-q13 and is inherited in a recessive manner. The metabolic anomaly results in the accumulation of glycogen in the liver and muscle.

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